Help your patients improve their oral health by completing this comprehensive program designed by TECH"

This Master's Degree in Oral Medicine is based on the student acquiring in-depth knowledge of different types of oral cavity and adjoining tissue lesions, both benign and malignant, as well as diagnostic and surgical techniques, correct treatment approaches, professional application and preventive usefulness.

It is a very useful branch, since it complements the rest of the specialties, being a fundamental pillar prior to any treatment, avoiding negligence due to the lack of knowledge and making it possible to detect and treat pathologies, which may potentially save a patient’s life.

This academic program is characterized by its dynamic methodology that intersperses clinical cases for the student to identify and associate the subject matter explained with its corresponding image, as well as questionnaires to evaluate their knowledge and test them, bringing it as close as possible to those situations that will be presented daily in the office, to be able to focus and manage them in a coordinated, efficient and planned way, all under the guidance of working professionals who will help during the learning process to achieve a complete education in all aspects.

It is based on promoting lasting and quality learning through schematically structured scientific information, always focusing on the main aspects of each pathology in order to be able to apply it in daily practice immediately after each unit.

The dentist will strengthen their personal confidence and decision-making abilities when practising, with special emphasis on diagnostic and preventive aspects, differentiating, according to specific characteristics, the different types of lesions so that their learning is fluid and effective.

As it is an online Master's Degree, the student is not hindered by fixed schedules or the need to move to a physical location, but can access the contents at any time of the day, balancing their professional or personal life with their academic life.

Only with adequate education will you learn the best way to advise your patients in Oral Medicine cases"

This Master's Degree in Oral Medicine contains the most complete and up to date scientific program on the market. The most important features include:

• More than 75 clinical cases presented by experts in Oral Medicine
• The graphic, schematic and practical contents of the course are designed to provide all the essential information required for professional practice
• Exercises where the self-assessment process can be carried out to improve learning
• Algorithm-based interactive learning system for decision making for the orally impaired patient
• Theoretical lessons, questions to the expert, debate forums on controversial topics, and individual reflection assignments
• Content that is accessible from any fixed or portable device with an Internet connection

This Master's Degree is the best investment you can make in the selection of a refresher program for two reasons: in addition to updating your knowledge in Oral Medicine, you will obtain a qualification from TECH Global University"

It includes, in its teaching staff, professionals belonging to the field of Oral Medicine, who contribute their work experience to this program, as well as leading specialists from prestigious societies and universities.

The multimedia content, developed with the latest educational technology, will provide the professional with situated and contextual learning, i.e., a simulated environment that will provide immersive training programmed to train in real situations.

This program is designed around Problem-Based Learning, whereby the professional must try to solve the different professional practice situations that arise throughout the program. To do so, the professional will be assisted by an innovative interactive video system created by renowned and experienced experts in Oral Medicine.

This Master's Degree offers training in simulated environments which provides an immersive learning experience designed to train for real-life situations"

This 100% online ’Master’s Degree will allow you to balance your studies with your professional work while increasing your knowledge in this field"

The structure of the contents has been designed by a team of professionals knowledgeable about the implications of the program in daily practice, aware of the current relevance of education in Oral Medicine and committed to quality teaching through new educational technologies.

We have the most complete and up-to-date programme on the market. We seek excellence and we want to help you achieve it too" 

Module 1. Oral Medicine and Diagnostic Methods

1.1. Pathology and Oral Medicine

1.1.1. In-Depth Oral Medicine
1.1.2. Relevant Figures
1.1.3. Oral Medicine Applied to Health Care Branches
1.1.4. Current Uses of Oral Medicine in Dentistry
1.1.5. Advances and Technology

1.2. Medical History

1.2.1. Medical History
1.2.2. Personal and Family History
1.2.3. Exploration
1.2.4. Diagnosis
1.2.5. Treatment Plan

1.3. Informed Consent

1.3.1. Origins and Fundamentals
1.3.2. Features
1.3.3. Applicable Exceptions
1.3.4. The Right to Information
1.3.5. The Right to Confidentiality

1.4. Legal Implications in Health Care

1.4.1. Origin and Fundamentals
1.4.2. Legal Principles Applied to Health Care
1.4.3. Obligations and Rights of the Professional
1.4.4. Legal Relevance of Medical Records
1.4.5. Relationship between Health and Administrative Management

1.5. Complementary Tests

1.5.1. Radiography
1.5.2. Nuclear Magnetic Resonance (NMR)
1.5.3. CT or CBCT
1.5.4. Electromyography
1.5.5. Sialometry
1.5.6. Ultrasound
1.5.7. Analytics
1.5.8. Urinalysis
1.5.9. Capillary Glycemia
1.5.10. INR
1.5.11. Exudates
1.5.12. FNA, Biopsy and Cytology
1.5.13. Mantoux Test
1.5.14. Breath Test
1.5.15. Endocrine Tests
1.5.16. Pulse Oximetry and Densimetry
1.5.17. Photography

1.6. Radiography

1.6.1. Intraoral X-Rays Types
1.6.2. Extraoral X-Rays Projections

1.7. Diagnostic Tests in Oral Medicine

1.7.1. Clinical Tests
1.7.2. Patch Test
1.7.3. Diagnostic Imaging
1.7.4. Contrast Diagnostics
1.7.5. Nuclear medicine
1.7.6. Culture Techniques
1.7.7. Immunological and Immunohistochemical Techniques

1.8. Biopsy

1.8.1. Fundamentals
1.8.2. Indications and Applications
1.8.3. Types and Procedures
1.8.4. Most Frequent Errors
1.8.5. Technical Contraindications for Biopsies

1.8.5.1. Materials
1.8.5.2. Incisional
1.8.5.3. Excisional
1.8.5.4. FNA
1.8.5.5. Cytology

1.9. Validity of Diagnostic Tests

1.9.1. Sensitivity
1.9.2. Specificity
1.9.3. Safety
1.9.4. Predictive Values
1.9.5. Accuracy
1.9.6. Precision

1.10. Research

1.10.1. Observation or Research?
1.10.2. Types of Studies
1.10.3. Systematic Reviews
1.10.4 Meta-analytical Study
1.10.5. Clinical Trials
1.10.6. Publication and Scientific Articles: Criteria

Module 2. Applied Anatomopathology and Elementary Lesions

2.1. Pathology Branches

2.1.1. General Pathology
2.1.2. Systemic Pathology
2.1.3. Molecular Pathology
2.1.4. Molecular Biology
2.1.5. Dental and Health Care Applications

2.2. Oral Mucosal Histopathology

2.2.1. Anatomy Recap
2.2.2. Histological Structure
2.2.3. Microscopic Elementary Lesions of the Oral Mucosa
2.2.4. Epithelial Tissue

2.2.4.1. Keratinized
2.2.4.2. Non-Keratinized

2.2.5. Epithelial Cell Junctions

2.2.5.1. Desmosome
2.2.5.2.  Hemidesmosomes
2.2.5.3. Others

2.3. Pathological Anatomy Fundamentals

2.3.1. Applications
2.3.2. Techniques
2.3.3. Study Method

2.3.3.1. Autopsy
2.3.3.2. Experimental Method

2.4. Functional Classification of Oral Mucosa

2.4.1. External Labial Mucosa
2.4.2. Lining Mucosa
2.4.3. Specialized Mucosa

2.5. Elementary Lesions

2.5.1. Features
2.5.2. Classification
2.5.3. Etiology
2.5.4. Chemical Agents

2.5.4.1. Chemical Burns: Substances and Drugs
2.5.4.2. Post-Anesthesia Necrosis
2.5.4.3. Secondary Drug Lesions

2.5.5. Physical Agents

2.5.5.1. Burns

2.5.5.1.1. Thermal
2.5.5.1.2. Electrical

2.5.6. Mechanical Agents

2.5.6.1. Alba Line
2.5.6.2. Frictional Hyperkeratosis
2.5.6.3. Leukoedema
2.5.6.4. Nibbling
2.5.6.5. Trauma
2.5.6.6. Ulcers

2.5.6.6.1. Decubitus
2.5.6.6.2. Traumatic

2.5.7. Allergic Oral Pathology

2.5.7.1. Angioedema
2.5.7.2. Allergic Contact Stomatitis
2.5.7.3. Anaphylactic Shock

2.5.8. Iatrogenesis

2.6. Solid Content Primary Lesions

2.6.1. Macula
2.6.2. Papule
2.6.3. Nodes
2.6.4. Bleb
2.6.5. Tuber
2.6.6. Rubber
2.6.7. Keratosis
2.6.8. Tumors

2.7. Liquid Content Primary Lesions

2.7.1. Phylctena
2.7.2. Gall Bladder
2.7.3. Blister
2.7.4. Pustules
2.7.5. Cyst

2.8. Secondary Lesions

2.8.1. Continuity Solution
2.8.2. Removable Residue
2.8.3. Restorative Processes

2.9. Staining

2.9.1. Oral Mucosa Dyschromia
2.9.2. Exogenous
2.9.3. Endogenous

2.10. Other Lesions

2.10.1. Sclerosis
2.10.2. Ulcers and Erosion
2.10.3. Lichenification
2.10.4. Intertrigo
2.10.5. Infiltration
2.10.6. Ocular Involvement

Module 3. Inflammatory and Infectious Oral Pathology

3.1. Bacterial Infections

3.1.1. Features
3.1.2. Scarlet Fever
3.1.3. Impetigo
3.1.4. Angular Cheilitis
3.1.5. Telangiectatic Granuloma
3.1.6. Cellulite

3.1.6.1. Acute
3.1.6.2. Chronic

3.1.7. Necrotizing Gingivitis
3.1.8. Gonococcal Pharyngitis
3.1.9. Syphilis

3.1.9.1. Primary
3.1.9.2. Secondary
3.1.9.3. Tertiary

3.1.10. TB
3.1.11. Leprosy
3.1.12. Actinomycosis
3.1.13. Gonorrhoea
3.1.14. Adenitis
3.1.15. Fistulas

3.2. Fungal Infections

3.2.1. Etiology
3.2.2. Classification

3.2.2.1. Thrush or Acute Pseudomembranous Candidiasis
3.2.2.2. Erythematous Candidiasis
3.2.2.3. Leukoplastic Candidiasis
3.2.2.4. Erythematous Candidiasis: Erosive Atrophic
3.2.2.5. Angular Cheilitis
3.2.2.6. Rhomboid Glossitis
3.2.2.7. Prosthetic Stomatitis
3.2.2.8. Deep Mucositis
3.2.2.9. Blastomycosis

3.3. Viral Infections

3.3.1. Characteristics and Treatment
3.3.2. Papillomas
3.3.3. Warts
3.3.4. Focal Epithelial Hyperplasia
3.3.5. Condyloma Acuminatum
3.3.6. Oral Condylomatosis
3.3.7. HSV Recurrent Herpes Labialis
3.3.8. Herpetic Primoinfection, Varicella Zoster and Herpes Zoster
3.3.9. Molluscum Contagiosum
3.3.10. Coxsackie
3.3.11. Herpangina
3.3.12. Hand-Foot-Mouth Disease
3.3.13. Paramyxovirus
3.3.14. Measles
3.3.15. CMV Mononucleosis
3.3.16. Epstein-Barr
3.3.17. Kawasaki Syndrome

3.4. Benign Exophytic Lesions

3.4.1. Etiology
3.4.2. Reactive Hyperplasia

3.4.2.1. Fibroepithelial Hyperplasia
3.4.2.2. Diapneusia
3.4.2.3. Papillary Palatine Hyperplasia
3.4.2.4. Fissured Granuloma
3.4.2.5. Fibrous Nodule
3.4.2.6. Reactive Granulomas
3.4.2.7. Peripheral Giant Cell Granuloma

3.4.3. Salivary Cysts

3.4.3.1. Caused by Retention
3.4.3.2. Caused by Extravasation

3.4.4. Benign Tumors

3.4.4.1. Epithelial
3.4.4.2. Connective

3.5. Connective Tissue Alterations

3.5.1. Sjögren's Syndrome
3.5.2. Lupus Erythematosus
3.5.3. Systemic Sclerosis
3.5.4. Rheumatoid Arthritis
3.5.5. Connective Tissue Tumors

3.5.5.1. Fibroma
3.5.5.2. Angioma

3.6. Maxillary and Mandibular Pathology

3.6.1. Features
3.6.2. Agnathia
3.6.3. Macrognathia
3.6.4. Micrognathia
3.6.5. Cleft Palate
3.6.6. Asymmetries
3.6.7. Treatment

3.7. Labial Pathology

3.7.1. Features
3.7.2. Fistulas and Labial Pits
3.7.3. Cleft Lip
3.7.4. Morsicatio Buccarum
3.7.5. Cheilitis

3.7.5.1. Cheilitis Simplex
3.7.5.2. Actinic Cheilitis
3.7.5.3. Allergic Contact Cheilitis
3.7.5.4. Cheilitis Glandularis
3.7.5.5. Exfoliative Cheilitis
3.7.5.6. Granulomatous Cheilitis
3.7.5.7. Macrocheilitis

3.7.6. Peutz-Jeghers Syndrome
3.7.7. Mucocele
3.7.8. Tumors and Pseudotumors

3.8. Lingual Pathology

3.8.1. Features
3.8.2. Hair Removal
3.8.3. Saburral Tongue
3.8.4. Macroglossia
3.8.5. Ankyloglossia
3.8.6. Median Rhomboidal Glossitis
3.8.7. Hairy Tongue
3.8.8. Fissured Tongue
3.8.9. Lingual Varicosities
3.8.10. Migratory Glossitis
3.8.11. Geographic Tongue
3.8.12. Cleft Tongue
3.8.13. Forked Tongue
3.8.14. Tumours
3.8.15. Motor Disturbances
3.8.16. Sensory Alterations

3.9. Blistering-Vesicular Diseases

3.9.1. Features and Types
3.9.2. Pemphigus

3.9.2.1. Vulgar
3.9.2.2. Erythematous
3.9.2.3. Foliaceous
3.9.2.4. Vegetans
3.9.2.5. Paraneoplastic

3.9.3. Pemphigoid

3.9.3.1. Cicatricial
3.9.3.2. Blistered

3.9.4. Linear IgA Dermatosis

3.9.4.1. Infantile
3.9.4.2. Adults

3.9.5. Exudative Erythema Multiforme

3.9.5.1. Features
3.9.5.2. Etiology and Predisposing Factors
3.9.5.3. Sevens-Johnson Syndrome
3.9.5.4. Toxic Epidermal Necrolysis
3.9.5.5. Evolution, Prognosis, and Treatment

3.9.6. Recurrent Aphthous Stomatitis (RAS)

3.9.6.1. Features
3.9.6.2. Etiology and Predisposing Factors
3.9.6.3. Major RAS
3.9.6.4. Minor RAS
3.9.6.5. Herpetiform Aphthous Stomatitis
3.9.6.6. Treatment

3.9.7. Associated Pathology and Syndromes

3.9.7.1. Celiac Disease
3.9.7.2. Crohn's Disease
3.9.7.3. Neutropenia
3.9.7.4. Behçet's Disease

3.10. Oral Lichen Planus

3.10.1. Etiology
3.10.2. Classification

3.10.2.1. Papular
3.10.2.2. Reticular
3.10.2.3. Atrophic
3.10.2.4. Erosive
3.10.2.5. Blistered
3.10.2.6. Plaque-type
3.10.2.7. Others

3.10.3. Diagnosis
3.10.4. Treatment
3.10.5. Dermatitis Herpetiformis

3.11. Nutritional Alterations

3.11.1. Metabolic Alterations

3.11.1.1. Amyloidosis
3.11.1.2. Lipoid Proteinosis
3.11.1.3. Fabry Disease

3.11.2. Vit A
3.11.3. Vit B2
3.11.4. Vit B3
3.11.5. Vit C
3.11.6. Folic Acid
3.11.7. Zinc

Module 4. Special Patients: Relation between Systemic Diseases and Oral Pathologies

4.1. Hematologic Alterations

4.1.1. Introduction
4.1.2. Red Series Diseases

4.1.2.1. Anaemia
4.1.2.2. Polyglobulia

4.1.3. White Blood Cell Disorders

4.1.3.1. Transplant Recipients: Before and After
4.1.3.2. HIV
4.1.3.3. Oncology Patients
4.1.3.4. Immunosuppressive Therapy for Autoimmune Pathology

4.1.4. Coagulation Disorders

4.1.4.1. Pharmacological Anticoagulants
4.1.4.2. Haemophilia
4.1.4.3. Secondary to Other Pathologies

4.1.5. Langerhans Cell Histiocytosis

4.2. Endocrine Disorders

4.2.1. Introduction
4.2.2. Glands and Organs

4.2.2.1. Adrenal Gland
4.2.2.2. Pancreas
4.2.2.3. Kidneys
4.2.2.4. Brain
4.2.2.5. Genital System

4.2.3. Endocrine-Metabolic Pathology
4.2.4. Dialysis
4.2.5. Adrenal Insufficiency

4.2.5.1. Primary: Addison's Disease
4.2.5.2. Secondary:

4.2.6. Diabetes Mellitus

4.2.6.1. Types
4.2.6.2. Protocol
4.2.6.3. Hemochromatosis or Bronzed Diabetes

4.2.7. Thyroid Pathology

4.2.7.1. Hyperthyroidism
4.2.7.2. Hypothyroidism
4.2.7.3. Tumours

4.3. Digestive Alterations

4.3.1. Anatomy
4.3.2. Crohn's Disease
4.3.3. Ulcerative Colitis
4.3.4. Gastroesophageal Reflux
4.3.5. Hepatopathy or Liver Disease
4.3.6. Uremic Stomatitis
4.3.7. Related Oral Pathology and Treatment
4.3.8. Prevention

4.4. Pulmonary Alterations

4.4.1. Anatomy
4.4.2. Types and Diagnostic Tests
4.4.3. COPD
4.4.4. Wegner Disease
4.4.5. Sarcoidosis
4.4.6. Related Oral Pathology
4.4.7. Action Protocol

4.5. Cardiovascular Problems

4.5.1. Circulatory System
4.5.2. Valvulopathies
4.5.3. Cardiomyopathies
4.5.4. Pericardiopathies
4.5.5. Aorta Diseases
4.5.6. Hypertension
4.5.7. Action Protocol

4.5.7.1. Antibiotic Prophylaxis
4.5.7.2. Anesthesia

4.6. Neurological Alterations:

4.6.1. Nervous system

4.6.1.1. Central
4.6.1.2. Peripheral

4.6.2. Cerebrovascular Diseases
4.6.3. Cerebrovascular Accidents

4.6.3.1. Hemorrhagic
4.6.3.2. Ischemic

4.6.4. Epilepsy
4.6.5. Related Oral Pathology
4.6.6. Prevention
4.6.7. Action Protocol

4.7. Dependent Patients

4.7.1. Types
4.7.2. Geriatric Patient
4.7.3. Addicted Patients

4.7.3.1. Tobacco
4.7.3.2. Alcohol
4.7.3.3. Drugs
4.7.3.4. Drugs:
4.7.3.5. Unhealthy Habits

4.7.4. Disability

4.7.4.1. Intellectual
4.7.4.2. Sensory
4.7.4.3. Motor

4.7.5. Related Oral Pathology
4.7.6. Prevention
4.7.7. Action Protocol

4.8. Pregnancy

4.8.1. Definition
4.8.2. Nursing
4.8.3. Related Oral Pathology

4.8.3.1. Gingivitis
4.8.3.2. Pyogenic Granuloma
4.8.3.3. Cavities
4.8.3.4. Periodontal Disease

4.8.4. Dental Emergencies
4.8.5. Prevention
4.8.6. Action Protocol

4.9. Emergencies

4.9.1. Cognitive Alterations
4.9.2. Respiratory Alterations
4.9.3. Cardiac Alterations
4.9.4. Allergies
4.9.5. Thoracic or Abdominal Pain
4.9.6. Anaphylactic Shock
4.9.7. Action Protocol

4.10. Oncology Patients

4.10.1. Definition
4.10.2. Types of Treatment

4.10.2.1. Radiotherapy
4.10.2.2. Chemotherapy
4.10.2.3. Brachytherapy
4.10.2.4. Surgical

4.10.3. Oncologic Treatment Phases
4.10.4. Related Oral Pathology
4.10.5. Prevention
4.10.6. Action Protocol

Module 5. Salivary Gland and TMJ Pathology

5.1. Saliva and Salivary Gland Anatomy

5.1.1. Composition
5.1.2. Functions
5.1.3. Saliva Flow Variations
5.1.4. Applications and Diagnostic Use
5.1.5. Salivary Gland Anatomy Recap

5.1.5.1. Parotid Gland
5.1.5.2. Sublingual Gland
5.1.5.3. Submaxillary Gland
5.1.5.4. Minor or Accessory Salivary Glands

5.2. Salivary Gland Malformations and Pathologies

5.2.1. Exploration
5.2.2. Fistulas
5.2.3. Stafne Cavity
5.2.4. Pathologies and Causes
5.2.5. Diagnostic tests

5.2.5.1. Radiological Diagnosis
5.2.5.2. Sialography Uses
5.2.5.3. Gammagraphy Uses

5.2.6. Complementary Tests
5.2.7. Serologic Test

5.3. Sialadenitis

5.3.1. Features
5.3.2. Pathologies

5.3.2.1. Bacterial Suppurative
5.3.2.2. Viral

5.3.2.2.1. Epidemic Mumps
5.3.2.2.2. Cytomegalic Mumps

5.3.3. Chronicle

5.3.3.1. Bacterial

5.3.3.1.1. Tuberculous
5.3.3.1.2. Actinomycosis
5.3.3.1.3. Syphilitic

5.3.3.2. Allergic/Toxic
5.3.3.3. Post Radiotherapy
5.3.3.4. Sclerosant
5.3.3.5. Recurrent (Juvenile)

5.4. Sialolithiasis

5.4.1. Features
5.4.2. Types

5.4.2.1. Pathologies
5.4.2.2. Chronicle

5.4.3. Mucocele
5.4.4. Garel's Hernia
5.4.5. Salivary Colic
5.4.6. Sialodochitis
5.4.7. Cannula
5.4.8. Treatment

5.5. Sialoadenosis

5.5.1. Features
5.5.2. Sarcoidosis
5.5.3. Cystic fibrosis
5.5.4. Sjögren's Syndrome

5.6. Tumor Pathology and Other Involvements

5.6.1. Features
5.6.2. Retention Cysts
5.6.3. Tumours
5.6.4. Frey Syndrome
5.6.5. Necrotizing Sialometaplasia

5.7. TMJ Anatomy

5.7.1. Bone Anatomy
5.7.2. Muscular Anatomy
5.7.3. Ligaments
5.7.4. Buttresses
5.7.5. Disks

5.8. TMJ Etiopathogenesis

5.8.1. Endocrine/Rheumatic Factors
5.8.2. Trauma
5.8.3. Psychosocial Factors

5.9. Pathologies. Classification

5.9.1. Congenital and Developmental Disorders
5.9.2. Condylar Pathology
5.9.3. Masticatory Muscle Disorders
5.9.4. Bone Pathology

5.9.4.1. Ankylosis
5.9.4.2. Arthritis

5.9.5. Tumorous Pathology

5.10. Exploration and Treatment

5.10.1. Clinical Examination
5.10.2. Diagnostic tests

5.10.2.1. Ultrasound
5.10.2.2. Arthroscopy
5.10.2.3. Resonance
5.10.2.4. CAT
5.10.2.5. Open Mouth/Closed Mouth X-ray
5.10.2.6. Osteoprotegerin (OPG)

5.10.3. Treatment

5.10.3.1. Unloading Splint
5.10.3.2. Occlusal Adjustment

5.10.3.2.1. Selective Grinding
5.10.3.2.2. Orthodontics

5.10.3.3. Pharmacological
5.10.3.4. Botulinum toxin
5.10.3.5. Physiotherapy
5.10.3.6. Surgical

Module 6. Bone Lesions and Maxillary Cysts

6.1. General Information on Bone Tissue

6.1.1. Bone Tissue and Histology
6.1.2. Transformation and Remodeling

6.1.2.1. Systemic Factors
6.1.2.2. Local Factors

6.1.3. Concepts and Terminology

6.1.3.1. Hyperplasia
6.1.3.2. Dysplasia
6.1.3.3. Neoplasia

6.2. Etiopathogenesis and Classification

6.2.1. Classification
6.2.2. Predisposing Factors
6.2.3. Etiology
6.2.4. Diagnostic Tests

6.3. Bone Pathology

6.3.1. Osteoporosis
6.3.2. Osteomalacia
6.3.3. Osteoclerosis
6.3.4. Fibrous Dysplasia
6.3.5. Parathyroid Osteosis
6.3.6. Lymphomas
6.3.7. Myelomas

6.4. Maxillary Bone Infections

6.4.1. Periodontitis
6.4.2. Cellulite

6.4.2.1. Pathologies
6.4.2.2. Chronic

6.4.3. Fistulae

6.4.3.1. Acquired
6.4.3.2. Chronic

6.4.4. Osteitis
6.4.5. Osteomyelitis
6.4.6. Osteoperiostitis

6.5. Other Bone Pathologies

6.5.1  Osteogenesis Imperfecta
6.5.2. Osteonecrosis
6.5.3. Osteoradionecrosis
6.5.4. Bisphosphonates

6.5.4.1. Features
6.5.4.2. Clinical Management

6.6. Developmental Epithelial Odontogenic Cysts

6.6.1. Infant Gingival Cyst or Epstein Pearls
6.6.2. Primordial Cyst
6.6.3. Dentigerous or Follicular Cysts
6.6.4. Eruption Cyst
6.6.5. Lateral Periodontal Cyst
6.6.6. Adult Gingival Cyst
6.6.7. Glandular Odontogenic Cyst
6.6.8. Odontogenic Keratocyst

6.7. Non-Odontogenic Developmental Epithelial Cysts

6.7.1. Nasopalatine Duct Cyst
6.7.2. Nasolabial Cyst
6.7.3. Globulomaxillary Cyst
6.7.4. Median Alverolary, Palatine and Mandibular Cysts
6.7.5. Differential Diagnosis

6.8. Inflammatory Epithelial Cysts

6.8.1. Radicular Cyst

6.8.1.1. Apical and Lateral Cyst
6.8.1.2. Residual Cyst

6.8.2. Paradental Cyst
6.8.3. Differential Diagnosis

6.9. Non-Neoplastic Bone Lesions or Pseudocysts

6.9.1. Solitary Bone Cyst
6.9.2. Aneurysmal Bone Cyst
6.9.3. Differential Diagnosis

6.10. Osteofibrous Diseases

6.10.1. Maxillary Fibrous Dysplasia
6.10.2. Cemento-Osseous Dysplasias

6.10.2.1. Periapical Cemento-Osseous Dysplasia
6.10.2.2. Florid Cemento-Osseous Dysplasia

6.10.3. Cherubism
6.10.4. Giant Cell Central Granuloma
6.10.5. Albright Syndrome
6.10.6. Paget’s Disease
6.10.7. Caffey’s Disease
6.10.8. Histiocytosis X
6.10.9. Syndrome Syndrome
6.10.10. Osteogenic Neoplasms

Module 7. Benign Tumors

7.1. Etiopathogenesis and Classification

7.1.1. Histology
7.1.2. Classification
7.1.3. Predisposing Factors
7.1.4. Etiology

7.2. Connective Tissue and Muscular Tumors

7.2.1. Features
7.2.2. Fibroma
7.2.3. Myxoma
7.2.4. Xanthoma Verruciformis
7.2.5. Nodular Fasciitis
7.2.6. Fibrous Hyperplasia
7.2.7. Tuberosity Bilateral Fibrous Hyperplasia
7.2.8. Fibrous Gingival Epulis
7.2.9. Cracked Epulis
7.2.10. Peripheral Giant Cell Granuloma (PGCG)
7.2.11. Myomas
7.2.12. Rhabdomyomas
7.2.13. Treatment

7.3. Vascular Tumours

7.3.1. Features
7.3.2. Hemangioma
7.3.3. Lymphangioma
7.3.4. Hemangioendothelioma
7.3.5. Features
7.3.6. Hemangiopericytoma
7.3.7. Glomus tumour
7.3.8. Pyogenic Granuloma
7.3.9. Pregnancy Epulis
7.3.10. Action Protocol

7.4. Neurogenic Tumors

7.4.1. Features
7.4.2. Neuromas

7.4.2.1. Traumatic
7.4.2.2. Neurofibromas
7.4.2.3. Von Recklinghausen Disease

7.4.3. Neurofibromas
7.4.4. Schwannoma
7.4.5. Action Protocol

7.5. Adipose Lineage Tumors

7.5.1. Features
7.5.2. Lipoma
7.5.3. Fordyce Granules
7.5.4. Superficial Abscesses
7.5.5. Differential Diagnosis
7.5.6. Treatment

7.6. Osteoforming Tumors

7.6.1. Torus

7.6.1.1. Mandibular
7.6.1.2. Palatal

7.6.2. Central and Peripheral Osteoma
7.6.3. Osteoma Osteoid
7.6.4. Osteoblastoma
7.6.5. Chondroma
7.6.6. Osteochondroma
7.6.7. Condroblastoma
7.6.8. Ossifying Fibroma

7.7. Non-Osteoforming Tumors

7.7.1. Fibrous Tumors

7.7.1.1. Non-Specific Fibroma
7.7.1.2. Chondromyxoid Fibroma
7.7.1.3. Desmoplastic Fibroma

7.7.2. Giant Cell Tumor

7.7.2.1. PGCG
7.7.2.2. Giant Cell Tumor

7.8. Ectomesenchymal with or without Odontogenic Epithelium Inclusion

7.8.1. Odontogenic Fibroma
7.8.2. Myxoma
7.8.3. Benign Cementoblastoma
7.8.4. Cemento-Ossifying Fibroma

7.9. Benign Odontogenic Tumors of Odontogenic Epithelium without Odontogenic Ectomesenchyma

7.9.1. Ameloblastomas
7.9.2. Calcifying Odontogenic Tumor or Pindborg's Tumor
7.9.3. Adenomatoid Squamous
7.9.4. Adenomatoid OT
7.9.5. Keratocystic OT

7.10. Benign Odontogenic Tumors of Odontogenic Epithelium without Odontogenic Ectomesenchyma

7.10.1. Ameloblastic Fibroma
7.10.2. Ameloblastic Fibrodentinoma (Dentinoma)
7.10.3. Odontoameloblastoma
7.10.4. Adenomatoid Odontogenic Tumor
7.10.5. Calcifying Odontogenic Tumor
7.10.6. Complex and Composite Odontoma
7.10.7. Calcifying Cystic Odontogenic Tumor or Gorlin's Cyst

Module 8. White and Premalignant Lesions

8.1. White Lesions

8.1.1. Classification

8.1.1.1. Hereditary Disorders
8.1.1.2. Reactive Lesions
8.1.1.3. Immunological Basis
8.1.1.4. Infectious Origin
8.1.1.5. Miscellaneous

8.1.2. Clinical Management

8.2. Premalignant Lesions

8.2.1. Concept of Premalignant Lesion
8.2.2. Histological Level
8.2.3. Classification
8.2.4. Predisposing Factors to Malignancy
8.2.5. Clinical Management

8.3. Leukoplakia

8.3.1. Features
8.3.2. Predisposing Factors
8.3.3. Etiology
8.3.4. Localization
8.3.5. Types

8.3.5.1. Homogeneous
8.3.5.2. Non-Homogeneous

8.3.5.2.1. Erythroleukoplakia
8.3.5.2.2. Nodular
8.3.5.2.3. Exophytic

8.3.5.2.3.1. Verrucose
8.3.5.2.3.2. Proliferative Verrucosa

8.3.6. Pathologic Anatomy/Pathogenesis

8.3.6.1. Stages
8.3.6.2. Dysplasia

8.3.7. Diagnosis
8.3.8. Treatment
8.3.9. Prognosis

8.4. Erythroplakia

8.4.1. Features
8.4.2. Predisposing Factors
8.4.3. Etiology
8.4.4. Localisation
8.4.5. Types

8.4.5.1. Homogeneous
8.4.5.2. Non-Homogeneous
8.4.5.3. Erythroleukoplakia

8.4.6. Diagnosis
8.4.7. Treatment
8.4.8. Prognosis

8.5. Actinic Cheilitis

8.5.1. Features
8.5.2. Predisposing Factors
8.5.3. Etiology
8.5.4. Treatment
8.5.5. Prognosis

8.6. Melanic Alterations

8.6.1. Features
8.6.2. Etiology
8.6.3. Diagnosis
8.6.4. Nevi
8.6.5. Pigmentary Nevus

8.6.5.1. Lentigo
8.6.5.2. Melanocytic Nevus
8.6.5.3. Acquired Melanocytic Nevus

8.6.5.3.1. Junctional or Union Nevus
8.6.5.3.2. Composite Nevus
8.6.5.3.3. Intradermal Nevus

8.6.6. Organoid Nevus

8.6.6.1. Epithelial
8.6.6.2. Conjunctive
8.6.6.3. Vascular

8.6.7. Prevention
8.6.8. Treatment

8.7. Submucosal Oral Fibrosis

8.7.1. Features
8.7.2. Predisposing Factors
8.7.3. Etiology
8.7.4. Treatment

8.8. Xeroderma Pigmentosum

8.8.1. Features
8.8.2. Predisposing Factors
8.8.3. Etiology
8.8.4. Treatment

8.9. Plummer Vinson Disease

8.9.1. Features
8.9.2. Predisposing Factors
8.9.3. Etiology
8.9.4. Treatment

8.10. Dyskeratosis Congenita

8.10.1. Features
8.10.2. Predisposing Factors
8.10.3. Etiology
8.10.4. Treatment

8.11. Epidermolysis Bullosa

8.11.1. Features
8.11.2. Predisposing Factors
8.11.3. Etiology
8.11.4. Treatment

Module 9. Oral Cancer and Malignant Tumors

9.1. Etiopathogenesis and Classification

9.1.1. Histology
9.1.2. Classification
9.1.3. Predisposing Factors
9.1.4. Etiology
9.1.5. Prevalence

9.2. Malignant Odontogenic Tumors: Odontogenic Carcinomas

9.2.1. Malignant Ameloblastoma
9.2.2. Primary Intraosseous Carcinoma
9.2.3. Sclerosing Odontogenic Carcinoma
9.2.4. OC Clear Cells
9.2.5. OC Ghost Cell OC
9.2.6. Odontogenic Cysts Presenting Malignant Changes

9.3. Malignant Odontogenic Tumors: Odontogenic Sarcoma

9.3.1. Ameloblastic Fibrosarcoma
9.3.2. Ameloblastic Fibrodentinosarcoma and Ameloblastic Fibro-Odontosarcoma

9.3.2.1. Odontogenic Carcinosarcoma

9.4. Squamous Cell Oral Carcinoma

9.4.1. Features
9.4.2. Etiology
9.4.3. Histology
9.4.4. Diagnosis
9.4.5. Prevention
9.4.6. Treatment
9.4.7. Prognosis
9.4.8. Evolution

9.5. Verrucous Carcinoma

9.5.1. Features
9.5.2. Etiology
9.5.3. Diagnosis
9.5.4. Prevention
9.5.5. Treatment
9.5.6. Prognosis
9.5.7. Evolution

9.6. Adenocarcinoma

9.6.1. Features
9.6.2. Etiology
9.6.3. Diagnosis
9.6.4. Classification and Types
9.6.5. Prevention
9.6.6. Treatment
9.6.7. Prognosis
9.6.8. Evolution

9.7. Oral Melanoma

9.7.1. Features
9.7.2. Classification
9.7.3. Etiology
9.7.4. Diagnosis
9.7.5. Prevention
9.7.6. Treatment
9.7.7. Prognosis
9.7.8. Evolution

9.8. Lymphatic Disorders

9.8.1. Features
9.8.2. Etiology
9.8.3. Diagnosis
9.8.4. Classification and Types
9.8.5. Prevention
9.8.6. Treatment
9.8.7. Prognosis
9.8.8. Evolution

9.9. Sarcomas

9.9.1. Features
9.9.2. Etiology
9.9.3. Diagnosis
9.9.4. Classification and Types
9.9.5. Prevention
9.9.6. Treatment
9.9.7. Prognosis
9.9.8. Evolution

9.10. Minor Salivary Gland Neoplasms

9.10.1. Features
9.10.2. Etiology
9.10.3. Diagnosis
9.10.4. Prevention
9.10.5. Treatment
9.10.6. Prognosis
9.10.7. Evolution

Module 10. Neuropathologies

10.1. Features
10.2. Origin

10.2.1. Lobes and Involvements
10.2.2. Function Alterations
10.2.3. Predisposing Factors
10.2.4. Etiology

10.3. Pain

10.3.1. Nomenclature
10.3.2. Nerve Fibers

10.3.2.1. Types
10.3.2.2. Neurotransmitters

10.3.3. Pathophysiology of Pain
10.3.4. Types of Pain
10.3.5. Treatment

10.4. Neuralgia

10.4.1. Definition
10.4.2. Types
10.4.3. Classification
10.4.4. Cranial Nerves
10.4.5. Spinal Nerves
10.4.6. Diagnosis
10.4.7. Treatment
10.4.8. Others

10.4.8.1. Facial Hemiatrophy
10.4.8.2. Minor Neuralgia
10.4.8.3. Fibromyalgia
10.4.8.4. Myofascial Pain

10.5. Trigeminal Neuralgia

10.5.1. Features
10.5.2. Origin
10.5.3. Predisposing Factors
10.5.4. Etiology
10.5.5. Diagnosis
10.5.6. Treatment
10.5.7. Evolution

10.6. Glossopharyngeal Neuralgia

10.6.1. Features
10.6.2. Origin
10.6.3. Predisposing Factors
10.6.4. Etiology
10.6.5. Diagnosis
10.6.6. Treatment
10.6.7. Evolution

10.7. Headaches and Cephalalgias

10.7.1. Clinical Classification
10.7.2. Pathophysiology
10.7.3. Migraines. Vascular-Type Algia
10.7.4. Cluster Headache
10.7.5. Other Orofacial Pain

10.7.5.1. Burning Mouth Syndrome
10.7.5.2. Atypical Facial Algia
10.7.5.3. Pterygoides Hamulus Syndrome
10.7.5.4. Pterygoid Process Syndrome

10.7.6. Palliative Techniques for Pain

10.8. Burning Mouth Syndrome

10.8.1. Features
10.8.2. Origin
10.8.3. Predisposing Factors
10.8.4. Etiology
10.8.5. Diagnosis
10.8.6. Treatment
10.8.7. Evolution

10.9. Facial Paralysis

10.9.1. Etiology

10.9.1.1. Pathology
10.9.1.2. Traumatic
10.9.1.3. Congenital
10.9.1.4. Idiopathic
10.9.1.5. Iatrogenic

10.9.2. Types

10.9.2.1. Central Facial Paralysis
10.9.2.2. Peripheral Facial Paralysis

10.9.3. Treatment
10.9.4. Miscellaneous

10.9.4.1. Guillain-Barré Syndrome
10.9.4.2. Paget’s Disease
10.9.4.3. Melkersson-Rosenthal Syndrome
10.9.4.4. Myofascial Syndrome
10.9.4.5. Lupus
10.9.4.6. ALS
10.9.4.7. Diabetic Neuropathy

10.10. Bell’s Palsy

10.10.1. Features
10.10.2. Origin
10.10.3. Predisposing Factors
10.10.4. Etiology
10.10.5. Diagnosis
10.10.6. Treatment
10.10.7. Evolution

10.11. Ramsay Hunt Syndrome

10.11.1. Features
10.11.2. Origin
10.11.3. Predisposing Factors
10.11.4. Etiology
10.11.5. Diagnosis
10.11.6. Treatment
10.11.7. Evolution

A unique, key, and decisive training experience to boost your professional development”