Thanks to this online program you will be able to update your knowledge on the main pathologies of the scalp and their treatments" 

Scientific studies, the use of new technologies and innovation in intervention methods have given a considerable boost to hair transplants. In this process, the role of medical professionals has undoubtedly been key, since their knowledge and technical skills have allowed surgical treatments to achieve great acceptance among the population and to move away from the taboo and negative image of undergoing this aesthetic therapy. 

At the same time, the boom in recent years of these treatments has led to the opening of more specialized clinics in the world, where the profiles of highly qualified surgeons, trichologists, dermatologists or anesthesiologists are increasingly in demand. For all these reasons, this Master’s Degree provides the specialist with a university education that will allow them to update their knowledge throughout the 12 months of its duration. The multimedia teaching material will lead the students to deepen their knowledge of the main hair-related diseases, dedicating a space to androgenic alopecia, cosmetic treatments and FUSS and FUE techniques.

In addition, simulations of clinical cases provided by the expert teaching staff will serve to bring students closer to real situations, helping them to broaden their skills. The professional has before them a program with a theoretical-practical approach oriented to offer the most recent knowledge and scientific rigor in the field of Hair Transplantation.

An advanced and intensive university program, but with the convenience of being able to take it exclusively online, without fixed class schedules. Thus, TECH favors that the medical professional can take a quality education compatible with their work responsibilities. Students only need a computer, tablet or cell phone to connect to the virtual platform where the syllabus is stored. Enriched content is available in full from day one, which gives students the freedom to distribute the course load according to their needs. The professional is, therefore, provided with flexible teaching that is at the academic forefront. 

A Master’s Degree that gives you the opportunity to access the latest content on advances in cloning and pharmacology for the resolution of patients' trichological problems" 

This Master’s Degree in Hair Transplantation contains the most complete and up-to-date scientific program on the market today, with the following notable features:

• Practical cases presented by experts in hair surgery
• The latest developments in Hair Surgery, with special and dedicated attention to the innovative methodologies
• Practical exercises where the self-evaluation process can be carried out to improve learning
• Graphic contents, diagrams, and practical cases that gather scientific and practical information on the disciplines that are essential for professional hair surgery practice
• Theoretical lessons, questions to experts and/or tutors, discussion forums on controversial issues and individual reflection work throughout the progam
• Content that is accessible from any, fixed or portable device, with an internet connection

A Master’s Degree that will give you 24-hour access to scientific and exhaustive content on Hair Transplantation" 

The program’s teaching staff includes professionals from sector who contribute their work experience to this program, as well as renowned specialists from leading societies and prestigious universities.

The multimedia content, developed with the latest educational technology, will provide the professional with situated and contextual learning, i.e., a simulated environment that will provide immersive training programmed to train in real situations.

This program is designed around Problem-Based Learning, whereby the professional must try to solve the different professional practice situations that arise throughout the program. For this purpose, the student will be assisted by an innovative interactive video system created by renowned and experienced experts. 

A 100% online, flexible educational program that allows you to distribute the course load according to your needs"

Deepen your knowledge of androgenetic alopecia and the AGA diagnostic study with this online program"

The syllabus of the Master’s Degree is structured as a comprehensive tour through each and every one of the concepts required to understand and work in this field. With an approach focused on practical application that will allow the student to grow as a professional from the first moment of specialization.

A comprehensive syllabus focused on acquiring knowledge and converting it into real skills, created to propel you to excellence"

Module 1. Hair Anatomy and Physiology

1.1. Normal Hair Follicle: Structure

1.1.1. Parts of the Hair Follicle

1.1.1.1. Upper Part

1.1.1.1.1. Follicular Ostium
1.1.1.1.2. Infundibulum
1.1.1.1.3. Isthmus

1.1.1.2. Lower Part

1.1.1.2.1. Erector Muscle Insertion
1.1.1.2.2. Hair Bulb: Melanocytes and Matrix Cells
1.1.1.2.3. Sebaceous Glands

1.1.2. Histology of the Hair Follicle
1.1.3. Mature Hair Follicle

1.1.3.1. Hair Shaft: Medulla, Cortex and Cuticle
1.1.3.2. Inner Root Sheath: Cuticle, Huxley's Layer and Henle's Layer
1.1.3.3. Outer Root Sheath
1.1.3.4. Vitreous Layer
1.1.3.5. Outer Fibrous Layer

1.1.4. Cellular Composition of the Hair Follicle

1.1.4.1. Stem Cells
1.1.4.2. Keratinocytes
1.1.4.3. Melanocytes
1.1.4.4. Neurons
1.1.4.5. Blood Vessels
1.1.4.6. Mastocytic Cell Precursor Derivatives
1.1.4.7. Immune Cells
1.1.4.8. Neuronal Stem Cells

1.2. Histopathological Signs of Non-Scarring Alopecias

1.2.1. Androgenetic Alopecia

1.2.1.1. Hair Follicle Miniaturization
1.2.1.2. Sebaceous Pseudohyperplasia

1.2.2. Telogen Effluvium 

1.2.2.1. Predominance of Hair Follicles in Telogen Phase
1.2.2.2. Absence of Significant Histopathological Changes

1.2.3. Alopecia Areata

1.2.3.1. Peri- and Intrabulbar Lymphocytic Infiltrate (Honeycomb)
1.2.3.2. Several Follicles of the Biopsy in the Same Evolutionary Phase
1.2.3.3. Reversal of the Anagen-Telogen Relationship

1.2.4. Syphilitic Alopecia

1.2.4.1. Abundance of Plasma Cells in the Inflammatory Infiltrate
1.2.4.2. Presence of Treponema Pallidum with HI stains

1.2.5. Trichotillomania

1.2.5.1. Absence of Peribulbar Inflammatory Infiltrate
1.2.5.2. Trichomalacia
1.2.5.3. Incontinentia Pigmenti
1.2.5.4. Intra and Perifollicular Hemorrhages

1.2.6. Traction Alopecia

1.2.6.1. Similar to Trichotillomania
1.2.6.2. Diminution of Terminal Hair Follicles

1.3. Anatomy of the Scalp

1.3.1. Scalp Layers (SCALP)

1.3.1.1. Skin

1.3.1.1.1. Epidermis and Dermis

1.3.1.1.1.1. Arrector Pili Muscles Smooth Muscle Fibers Innervation of the Sympathetic Nervous System Inserted into Hair follicle Goosebumps

1.3.1.1.2. Thick. Between 3mm (Vertex) to 8mm (Occipital)
1.3.1.1.3. Contains

1.3.1.1.3.1. Hair Follicles: Rich Innervation
1.3.1.1.3.2. Sebaceous Glands
1.3.1.1.3.3. Sweat Glands

1.3.1.2. Subcutaneous Tissue

1.3.1.2.1. Fat and Fibrous Septa
1.3.1.2.2. Arteries, Veins, Lymphatic Vessels, and Nerves

1.3.1.3. Aponeurosis

1.3.1.3.1. Musculoaponeurotic Scalp Layer
1.3.1.3.2. Epicranial Muscle
1.3.1.3.3. Traction Resistance

1.3.1.4. Lax Subcutaneous Tissue

1.3.1.4.1. Thin
1.3.1.4.2. Avascular/Emitting Veins
1.3.1.4.3. Infections

1.3.1.5. Pericranium/Periosteum

1.3.2. Skin Vascularisation

1.3.2.1. Deep Plexus: Vascular Trunks and Superficial Plexus
1.3.2.2. Subcutaneous Plexus, Cutaneous Plexus, Papillary Plexus, and Capillary Loop

1.4. Types of Hair

1.4.1. Lanugo

1.4.1.1. Fine
1.4.1.2. Non-Vascular

1.4.2. Vellus

1.4.2.1. Diameter and Length (30u/<1cm)
1.4.2.2. Depigmented and Non-Medullated
1.4.2.3. Vellus Hairs

1.4.3. Terminal Hair

1.4.3.1. Diameter and Length (60u/>1cm)
1.4.3.2. Pigmented and Medullated

1.4.4. According to Area of Growth

1.4.4.1. Asexual Hair
1.4.4.2. Ambosexual Hair
1.4.4.3. Sexual Hair

1.5. Chemical Composition of Hair

1.5.1. Protein (28%)

1.5.1.1. Keratin: Hard and Soft
1.5.1.2. Amino Acids: Cysteine, Histidine, Methionine and Tryptophan

1.5.2. Lipids (2%)
1.5.3. Trace Elements
1.5.4. Water (70%)
1.5.5. Pigments
1.5.6. Others

1.6. Hair Properties 

1.6.1. Permeability: Ability to Absorb Liquids

1.6.1.1. Changes in Length, Diameter, and Shape

1.6.2. Resistance: Ability to Withstand Traction

1.6.2.1. Determined by Its Structure and Chemical Composition
1.6.2.2. Relation to Sulfur

1.7. Hair Phases

1.7.1. Anagen phase

1.7.1.1. 4-6 years
1.7.1.2. Birth and Growth

1.7.2. Catagen Phase

1.7.2.1. 2-3 weeks
1.7.2.2. 1-2% of Hairs
1.7.2.3. Anabolic to Catabolic Transition

1.7.3. Telogen Phase

1.7.3.1. Resting Phase and Hair Shedding
1.7.3.2. 3- 4- months
1.7.3.3. 4-24% of Hairs
1.7.3.4. Increased In: Postpartum (Physiological) and (Pathological) such as Stress and Fever

1.8. Follicular Unit

1.8.1. Definition
1.8.2. Follicular Unit Density (UF/cm2)
1.8.3. Hair Density (Hairs/cm2)
1.8.4. Differences between Races

1.8.4.1. Asian vs. Colored vs. White Race

1.9. History of Hair Surgery

1.9.1. Early Beginnings. The Role of Japan in Hair Surgery

1.9.1.1. Eyebrows and Eyelashes

1.9.2. Beginning of its Development and Practice
1.9.3. The First Hair Transplant
1.9.4. The Evolution of Transplantation

1.9.4.1. The 1960s to the 1970s
1.9.4.2. The 1980s
1.9.4.3. 1984: Dr. John Headington: “Follicular Unit Transplantation”

1.9.5. Evolution of Surgical Techniques

1.9.5.1. Punch Grafts, Mini Grafts, Micro Grafts

1.9.6. Organisation

1.9.6.1. Scientific Societies
1.9.6.2. Specific Scientific Journals
1.9.6.3. Specific Textbooks

Module 2. Hair Disorders

2.1. Scalp Pathologies

2.1.1. Dermatosis

2.1.1.1. Dermatoses Affecting the Scalp

2.1.1.1.1. Seborrheic Dermatitis

2.1.1.1.1.1. Description and Origin
2.1.1.1.1.2. Phases of Seborrheic Dermatitis

2.1.1.2. Contact Dermatitis

2.1 1.2.1. Contact Irritant

2.1.1.2.1.1. Chemical Contact
2.1.1.2.1.2. Physical Contact (Allergens)

2.1.1.2.2. Photocontact or Photosensitive

2.1.1.2.2.1. Phototoxic
2.1.1.2.2.2. Photoallergic

2.1.1.3. Erosive-Pustular Dermatosis

2.1.2. Pityriasis

2.1.2.1. Pityriasis
2.1.2.2. Cosmetic Pityriasis
2.1.2.3. Pityriasis Simplex Capitis
2.1.2.4. Steatoid Pityriasis

2.1.3. Scalp Infections and Infestations

2.1.3.1. Superficial Folliculitis (Ostiofolliculitis)
2.1.3.2. Deep Folliculitis (Furunculosis and Carbuncles)

2.1.3.2.1. Folliculitis Decalvans

2.1.3.3. Keloid Folliculitis (Keloid Acne)
2.1.3.4. Candidiasis Folliculitis

2.1.4. Tinea Capitis

2.1.4.1. Non-Inflammatory Tinea (Anthropophilic Dermatophytes)
2.1.4.2. Inflammatory Tinea (By Zoophoric Dermatophytes)

2.1.5. Seborrheic Dermatoses, Description and Types

2.1.5.1. Real Seborrhea
2.1.5.2. Two-Layer Seborrhea
2.1.5.3. Apparent Seborrhea
2.1.5.4. Four-Layer Seborrhea
2.1.5.5. Lichen Planopilaris
2.1.5.6. Pediculosis
2.1.5.7. Capillary Psoriasis

2.1.5.7.1. Exclusive Capillary Involvement: Seborrheic Psoriasis
2.1.5.7.2. Plaques: Types

2.1.5.7.2.1. Isolated
2.1.5.7.2.2. Dispersed
2.1.5.7.2.3. Scarce

2.2. Bromhidrosis

2.2.1. Definition
2.2.2. Causes

2.2.2.1. Apocrine Sweating
2.2.2.2. Eccrine Sweating

2.2.3. Trigger Foods

2.2.3.1. Other Triggers

2.2.4. Symptoms
2.2.5. Diagnosis
2.2.6. Treatment

2.2.6.1. Botox
2.2.6.2. Liposuction
2.2.6.3. Surgery
2.2.6.4. Home Remedies

2.2.7. Complications

2.2.7.1. Trichomycosis
2.2.7.2. Erythrasma
2.2.7.3. Intertrigo
2.2.7.4. Diabetes Mellitus Type II
2.2.7.5. Obesity

2.3. Congenital Alopecia

2.3.1. Universal
2.3.2. Hereditary Hypotrichosis: Marie-Unna Type
2.3.3. Unclassified Types

2.3.3.1. Localized

2.3.3.1.1. Aplasia
2.3.3.1.2. Skin

2.3.3.2. Triangular Alopecia
2.3.3.3. Congenital Anonychia

2.3.4. Ectodermal Dysplasias

2.3.4.1. Hydrotic
2.3.4.2. Anhydrotic

2.3.5. Syndromes

2.3.5.1. Autosomal Recessive Conditions

2.3.5.1.1. Cockayne Syndrome
2.3.5.1.2. Werner Syndrome
2.3.5.1.3. Progeria
2.3.5.1.4. Rothmund-Thomson Syndrome
2.3.5.1.5. Seckel Syndrome
2.3.5.1.6. Menkes Syndrome
2.3.5.1.7. Marinesco Syndrome
2.3.5.1.8. Conradi Syndrome
2.3.5.1.9. Dyskeratosis Congenita
2.3.5.1.10. Cartilage-Hair Hypoplasia
2.3.5.1.11. Enteropathic Acrodematitis
2.3.5.1.12. Syndromes: Tricho-Rhino-Phalangeal
2.3.5.1.13. Homocystinuria
2.3.5.1.14. Lamellar Ichthyosis
2.3.5.1.15. Hartnut Disease
2.3.5.1.16. Citrulinemia
2.3.5.1.17. Trichorhinophalangeal Syndrome

2.3.6. Autosomal Dominant Conditions

2.3.6.1. Pachyonychia Congenita
2.3.6.2. Hallermann-Streiff Syndrome
2.3.6.3. Oculo-Dento-Digital Syndrome
2.3.6.4. Treacher-Collins Syndrome
2.3.6.5. Popliteal Membrane Syndrome

2.3.7. AX-Linked Dominant Disorders

2.3.7.1. Digital Orofacial Syndrome
2.3.7.2. Incontinentia Pigmenti
2.3.7.3. Focal Dermal Hypoplasia

2.3.8. AX Linked Recessive Disorders

2.3.8.1. Keratosis Follicularis Spinulosa Decalvans with Ophiasi

2.3.9. Chromosomal Aberrations

2.3.9.1. Down Syndrome - Trisomy 21
2.3.9.2. Trisomy A

2.4. Scarring Alopecia

2.4.1. Definition
2.4.2. Types

2.4.2.1. Caused by the Body Itself

2.4.2.1.1. Genetic Factors

2.4.2.2. Abnormalities

2.4.2.2.1. Folliculitis Decalvans
2.4.2.2.2. Keloid Acne
2.4.2.2.3. Lupus Erythematosus
2.4.2.2.4. Pustular Dermatosis
2.4.2.2.5. Lichen Planus
2.4.2.2.6. Frontal Fibrosing Alopecia (FFA)
2.4.2.2.7. Some Types of General Alopecia Areata

2.4.2.3. Acquired

2.4.2.3.1. Radiotherapy
2.4.2.3.2. Burns
2.4.2.3.3. Surgical

2.5. Other Classifications of Scarring Alopecia

2.5.1. Lymphocytic Infiltrate

2.5.1.1. Chronic Cutaneous Lupus Erythematosus
2.5.1.2. Follicular Lichen Planus
2.5.1.3. Pseudopelade of Brocq
2.5.1.4. Central Centrifugal Cicatricial Alopecia

2.5.2. Neutrophilic Infiltrate

2.5.2.1. Folliculitis Decalvans
2.5.2.2. Dissecting Cellulitis/Folliculitis

2.5.3. Mixed Infiltrate

2.5.3.1. Keloid Acne of the Nape
2.5.3.2. Varioliform Necrotic Acne
2.5.3.3. Erosive Pustular Dermatosis

2.5.4. Non-Specific Infiltrate

2.5.4.1. Scarring Alopecia in Final Stages

2.6. Non-Scarring Alopecia

2.6.1. Definition
2.6.2. Types

2.6.2.1. Androgenetic Alopecia
2.6.2.2. Traumatic or External Agent Alopecia

2.6.2.2.1. Trichotillomania
2.6.2.2.2. Caused by Chemical Misuse
2.6.2.2.3. Traction Alopecia

2.6.2.3. Alopecia Areata

2.6.2.3.1. Common Areata
2.6.2.3.2. General Areata

2.6.2.4. Drug and Pharmaceutical Related Alopecia

2.6.2.4.1. Vitamin A
2.6.2.4.2. Anticoagulants
2.6.2.4.3. Mercury
2.6.2.4.4. Boric Acid
2.6.2.4.5. Beta-Blockers

2.6.2.5. Syphilitic Alopecia

2.6.2.5.1. Description
2.6.2.5.2. Features

2.6.2.6. Alopecia Caused by Systemic Diseases

2.6.2.6.1. Infectious
2.6.2.6.2. Endocrine
2.6.2.6.3. Nutritional Deficiency

2.6.2.7. Effluvia

2.6.3. Histopathological Signs

2.6.3.1. Androgenetic Alopecia

2.6.3.1.1. Hair Follicle Miniaturization
2.6.3.1.2. Sebaceous Pseudohyperplasia

2.6.3.2. Telogen Effluvium

2.6.3.2.1. Predominance of Hair Follicles in Telogen Phase
2.6.3.2.2. Absence of Significant Histopathological Changes

2.6.3.3. Alopecia Areata

2.6.3.3.1. Peri- and Intra-bulbar Lymphocytic Infiltrate (Honeycomb)
2.6.3.3.2. Several Follicles of the Biopsy in the Same Evolutionary Phase
2.6.3.3.3. Reversal of the Anagen-Telogen Ratio

2.6.3.4. Syphilitic Alopecia

2.6.3.4.1. Abundance of Plasma Cells in the Inflammatory Infiltrate
2.6.3.4.2. Presence of Treponema Pallidum with HI stains

2.6.3.5. Trichotillomania

2.6.3.5.1. Absence of Peribulbar Inflammatory Infiltrate
2.6.3.5.2. Trichomalacia
2.6.3.5.3. Incontinentia Pigmenti
2.6.3.5.4. Intra and Perifollicular Hemorrhages

2.6.3.6. Traction Alopecia

2.6.3.6.1. Similar to Trichotillomania
2.6.3.6.2. Diminution of Terminal Hair Follicles

2.7. Hypertrichosis

2.7.1. General

2.7.1.1. Primary or Congenital

2.7.1.1.1. Universal Hypertrichosis or Ambras Syndrome
2.7.1.1.2. Congenital Hypertrichosis Lanuginosa
2.7.1.1.3. Prepubertal Hypertrichosis
2.7.1.1.4. Acquired Hypertrichosis Lanuginosa

2.7.1.2. Secondary or Acquired

2.7.1.2.1. Caused by Drugs or Medication
2.7.1.2.2. Caused by Systemic Diseases

2.7.2. Localized

2.8. Hirsutism

2.8.1. Ovarian SAHA Syndrome
2.8.2. Adrenal SAHA Syndrome
2.8.3. SAHA Syndrome with Hyperprolactinemia
2.8.4. SOP
2.8.5. Hypophyseal Hirsutism
2.8.6. Drug Use
2.8.7. Liver Diseases

2.9. Hyperhydrosis

2.9.1. Definition
2.9.2. Diagnosis
2.9.3. Causes

2.9.3.1. Education
2.9.3.2. Diffuse

2.9.4. Treatment

2.9.4.1. Antiperspirants
2.9.4.2. Anticholinergics
2.9.4.3. Iontophoresis
2.9.4.4. Botox
2.9.4.5. Microwave Thermolysis

Module 3. Androgenetic Alopecia

3.1. Features

3.1.1. Evolutionary Development
3.1.2. Physiological or Non-Physiological
3.1.3. Mediated by Two Factors: Genetic and Androgenic

3.2. Evolution

3.2.1. Hamilton for Boys
3.2.2. Ludwig for Girls

3.3. Pathophysiology

3.3.1. Genetic Receptors of the Male Hormone
3.3.2. An Enzyme the 5alpha-Reductase
3.3.3. DHT

3.4. Men
3.5. Women

3.5.1. Physiology
3.5.2. Hormonal
3.5.3. Genetics
3.5.4. Study of the Hypothalamic-Pituitary-Pituitary-Adrenal-Ovarian Axis

3.6. Consequences
3.7. AGA Study: Inclusion in Therapeutic Algorithm

3.7.1. Clinical History with Oriented Anamnesis
3.7.2. Macro and Micro Exploration with Use of Dermatoscopes and Micro Cameras
3.7.3. Taking Photographs
3.7.4. Traction Test
3.7.5. Trichogram

3.7.5.1. Optical Microscope: 20-50 Hairs
3.7.5.2. Classification of Growth Phases: Anagen (85%), Catagen (1-2%) and Telogen (10-15%)
3.7.5.3. Daily Hair Loss
3.7.5.4. Features

3.7.6. Wood Light
3.7.7. Biopsy
3.7.8. Targeted Analysis
3.7.9. Diagnostic Approximation

3.7.9.1. Inclusion in Therapeutic Algorithm: Prevention of Baldness

3.7.10. According to Resolution

3.7.10.1. Easily Resolved: Seasonal or Cyclic, Androgenetic (MAGA and FAGA), Menopausal and Senile Effluvium
3.7.10.2. Potentially Resolvable: Pathogen Mediated 

3.7.10.2.1. Psychogenic due to Stress
3.7.10.2.2. By Traction and Trichotillomania
3.7.10.2.3. Deficiency (Dietary, Anemic, Vitamin Deficiency)
3.7.10.2.4. Chronic Effluvium
3.7.10.2.5. Hormonal/Androgenic
3.7.10.2.6. Thyrogenic
3.7.10.2.7. Immunogenic
3.7.10.2.8. Chemotherapy
3.7.10.2.9. By Collagenosis
3.7.10.2.10. Areata
3.7.10.2.11. Infectious (Bacterial, Mycotic, Syphilis)
3.7.10.2.12. More Common in Women: Multi-Factorial

3.7.10.3. Difficult to Resolve

3.7.10.3.1. Congenital Scarring
3.7.10.3.2. FFA
3.7.10.3.3. Physical
3.7.10.3.4. Infections
3.7.10.3.5. Tumours
3.7.10.3.6. Dermatosis (Lupus, Liquen, Psoriasis, etc.)

3.8. Treatment

3.8.1. Cosmetic

3.8.1.1. Cleanliness and Hygiene: Adequate Shampoo
3.8.1.2. Moisturizes, Nourishes and Repairs the Hair Shaft
3.8.1.3. Powders, Dyes, Volumizing Sprays and Special Hairstyles
3.8.1.4. Keratin Microfibers
3.8.1.5. Extensions and Prostheses

3.8.2. Dietetic: Balanced Diet

3.8.2.1. Aminoacids: L-Cysteine
3.8.2.2. Vitamins: B12, Biotin, Folic Acid, etc.
3.8.2.3. Trace Elements: Zinc, Fe, Se, etc.

3.8.3. Topical

3.8.3.1. Non-Specific

3.8.3.1.1. In Shampoos: Antifungal, Antipsoriatic, Keratolytic, etc. 
3.8.3.1.2. Creams, Lotions, Gels, etc.
3.8.3.1.3. Corticosteroids, Antibiotics, Seboregulators, etc. 

3.8.3.2. Specific

3.8.3.2.1. In Lotions or Foams
3.8.3.2.2. Spinolactone 3 
3.8.3.2.3. Canrenone 2%
3.8.3.2.4. Progesterone 0.025%
3.8.3.2.5. 17-Alpha-Estradiol 0.025-0.05%
3.8.3.2.6. Minoxidil 2-5%
3.8.3.2.7. Ac. Retinoic Acid 0.025-0.05%
3.8.3.2.8. Alpha-Tocopherol Nicotinate 5%

3.8.4. Local

3.8.4.1. Drug Dermoinfiltration

3.8.4.1.1. Roller
3.8.4.1.2. Dermojet
3.8.4.1.3. Hair Mesotherapy
3.8.4.1.4. Carboxytherapy

3.8.4.2. Micropigmentation
3.8.4.3. Biological Therapies: PRP and Stem Cells
3.8.4.4. Electrophysical Therapy

3.8.4.4.1. Transportation and Ionization
3.8.4.4.2. Infrared and Low Frequency Lasers

3.8.4.5. Hair Surgery

3.8.5. Systemic

3.8.5.1. Underlying Pathology

3.8.5.1.1. Anti-Fungals/Antibiotics, Thyroid, Anxiolytics, Corticosteroids

3.8.5.2. Androgenetic Factor (AGA)

3.8.5.2.1. Finasteride
3.8.5.2.2. Dutasteride
3.8.5.2.3. Oral Minoxidil

3.8.5.3. Of Androgenic Factor: Anti-Androgens of Origin

3.8.5.3.1. Central: Cyproterone with/without Estradiol
3.8.5.3.2. Peripheral: Spironolactone
3.8.5.3.3. Adrenal: Prednisone and Deflazacort

3.9. Specific Techniques

3.9.1. Mesoterapiacapillary
3.9.2. Micro Hair Transplants
3.9.3. Biological Therapies

3.9.3.1. Plasma
3.9.3.2. Stem Cells

Module 4. Physician Attendance/Consultation and Surgery

4.1. Diagnostic Medical Consultation

4.1.1. Exploration Methods

4.1.1.1. Visual
4.1.1.2. Optical Microscope
4.1.1.3. Digital Micro Cameras
4.1.1.4. Micrometer
4.1.1.5. Wood Light
4.1.1.6. Traction Tweezers
4.1.1.7. Cigarette Paper

4.1.2. Performing a Trichogram
4.1.3. Dermographic Study
4.1.4. Traction Test
4.1.5. Wood Light
4.1.6. Biopsy (If Required)
4.1.7. Specific Blood Test
4.1.8. Photography

4.2. Pre-Surgical Medical Consultation

4.2.1. Medical History of Interest
4.2.2. Photography
4.2.3. Medical Consent
4.2.4. Patient Expectations
4.2.5. Surgical Plan
4.2.6. Pre-Op Instructions
4.2.7. Confirmation of Surgical Suitability
4.2.8. Post-Op Instructions
4.2.9. Surgical Alternatives and Other Treatments

4.3. Hair Transplant Devices

4.3.1. Hair Grafting Tools for Extraction

4.3.1.1. Punch or Circular Scalpel
4.3.1.2. SAFE Systems
4.3.1.3. RotoCore
4.3.1.4. NeoGraft® Automated FUE and Implantation System

4.3.2. Hair Grafting Tools for Implantation

4.3.2.1. Implanter

4.3.2.1.1. Advantages
4.3.2.1.2. Disadvantages

4.3.2.2. Corneal Incisions

4.3.2.2.1. Advantages
4.3.2.2.2. Disadvantages

4.3.3. Instruments for Making Incisions

4.3.3.1. Sharp Points
4.3.3.2. Needle
4.3.3.3. Mini-Blades

4.3.4. Main Developments

4.3.4.1. Extractor Machine with 0.7 mm Punch
4.3.4.2. Special Blades for Cutting and Trimming Follicular Units with the Strip Technique
4.3.4.3. High-Resolution Microscope
4.3.4.4. 3-5x Magnifiers
4.3.4.5. 0.8-1.3 Angled Blades for 0.8 mm and 1 mm Implants
4.3.4.6. 0.8mm and 1mm Implanters

4.3.5. Automated Extraction Systems - Capillary Implantation

4.3.5.1. Automatic: Extraction
4.3.5.2. Semi-Automatic: Extraction and Implantation

4.4. Aesthetic Eyebrow Procedures

4.4.1. Indications
4.4.2. Diagnosis
4.4.3. Etiology Treatment
4.4.4. Most Common Procedures

4.4.4.1. Recreating the Eyebrow Curve
4.4.4.2. Hair Density Recovery
4.4.4.3. Correcting Drooping Eyebrows

4.4.5. Postoperative Care
4.4.6. Anesthesia in Capillary Surgery

4.4.6.1. Pre-Operative Study
4.4.6.2. EKG and Chest X-Ray
4.4.6.3. Pre-Medication
4.4.6.4. Oral Sedation Model

4.4.6.4.1. Stomach Protector
4.4.6.4.2. Oral Antiemetic
4.4.6.4.3. Oral Dormicum 7.5mg 1 Hour before Surgery

4.5. Intravenous Options

4.5.1. Venous Route
4.5.2. Antiemetics
4.5.3. Administer Dormicum (2mg) and Fentanest (50 mg)
4.5.4. Portable Pulse Oximetry without the Need for Cardiac Monitoring
4.5.5. Annexate and Naloxone Availability

4.6. Types of Anesthetics

4.6.1. Ester Type: Tetracaine, Chlorprocaine, Benzocaine, and Procaine
4.6.2. Amide Type: Lidocaine, Mepivacaine, Prilocaine, Bupivacaine, Ropivacaine, and Etidocaine

4.7. Factors Influencing Its Action

4.7.1. Its Anesthetic Potency Is Related to Liposolubility in a Directly Proportional Manner
4.7.2. Vasodilation Capability
4.7.3. Plasma Protein Binding
4.7.4. Addition of a Vasoconstrictor, such as Adrenaline or Phenylephrine, Increases the Effect
4.7.5. Adrenaline Dosage Should Not Exceed 250ng in Adults
4.7.6. Alkalinization Improves Diffusion and Promotes Latency
4.7.7. Solution Heating Improves Blocking
4.7.8. Complications

4.7.8.1. Local Anesthesia Allergy
4.7.8.2. Local Anesthesia Toxicity

4.8. Non-Medical Team

4.8.1. Nursing

4.8.1.1. Functions

4.8.1.1.1. Assisting the Physician in Surgery
4.8.1.1.2. Extracting Follicular Units
4.8.1.1.3. Graft Trimming and Cleaning
4.8.1.1.4. Implanting Micrografts

4.8.2. Capillary Technician

4.8.2.1. Functions

4.8.2.1.1. Assisting the Nurse
4.8.2.1.2. Preparing the Room
4.8.2.1.3. Graft Trimming and Cleaning
4.8.2.1.4. Implanting Micrografts
4.8.2.1.5. Sterilization and Cleaning of the Room and Equipment

4.9. Complications/Emergencies in Surgery
4.10. Post-Surgery Treatment

4.10.1. Post-Operative Medication
4.10.2. Keep the Graft Site Clean and Hydrated
4.10.3. Sleep in a Half-Seated Position (40º/45º)
4.10.4. Avoid Sun Exposure
4.10.5. Minimal Physical Exercise
4.10.6. Apply Cold to the Face

Module 5. Cosmetic Treatments/Hair Cosmetics

5.1. Hair Cosmetics Definition. Concept. Materials Used
5.2. Hair Prosthesis. Definition. Differences between Male and Female

5.2.1. Manufacturing Materials

5.2.1.1. On the Outside of the Prosthesis: Different Types of Human Hair and Synthetic Hair Inside of the Prosthesis: Tulle, Gauze, Mesh, Plasticized

5.2.2. Manufacturing Techniques

5.2.2.1. Choppy Hair
5.2.2.2. Woven Hair

5.2.3. Fastening Materials

5.2.3.1. Self-Adhesives
5.2.3.2. Glues or Adhesives
5.2.3.3. Stitched

5.2.4. Importance of Hair Prosthesis Maintenance

5.3. Micropigmentation

5.3.1. Micropigmentation Techniques

5.3.1.1. Capillary
5.3.1.2. Eyebrows
5.3.1.3. Beard

5.3.2. Aspects to Consider when Applying Hair Micropigmentation
5.3.3. Products Used in Hair Micropigmentation
5.3.4. Equipment Used in Hair Micropigmentation
5.3.5. Micropigmentation Preservation

5.4. Hair Fibers

5.4.1. Spray
5.4.2. Powder

5.5. Definition of Cosmetic Treatments

5.5.1. Limitations of Cosmetic Treatments

5.6. Cosmetic Penetration

5.6.1. Penetration Routes
5.6.2. Degrees of Penetration
5.6.3. Penetration Factors

5.7. General Composition of Cosmetics. Active Ingredients, Excipients, Coloring, Perfume, Preservatives, Correcting Agents

5.7.1. Active ingredients

5.7.1.1. Plant: Origin, Procurement, and Composition
5.7.1.2. Animal: Origin, Procurement, and Composition

5.7.1.2.1. Synthetic: Origin, Procurement, and Composition
5.7.1.2.2. Others: Vitamins and Trace Elements
5.7.1.2.3. Excipients
5.7.1.2.4. Dyes
5.7.1.2.5. Perfumes
5.7.1.2.6. Preservatives
5.7.1.2.7. Correctors

5.8. Cosmetic Used in Hair Treatments

5.8.1. Dry Hair Cosmetics
5.8.2. Oily and Seborrheic Cosmetics
5.8.3. Pityriasis Cosmetics
5.8.4. Alopecia Cosmetics

5.9. Cosmetic Forms of Hair Treatments

5.9.1. Shampoos
5.9.2. Lotions
5.9.3. Peeling and Nourishing Masks
5.9.4. Micronutrients

5.10. Appliances Used in Cosmetic Treatments

5.10.1. Electrotherapy
5.10.2. High Frequency
5.10.3. Phototherapy

5.10.3.1. Infrared
5.10.3.2. Ultraviolet
5.10.3.3. Cosmetic Laser

5.10.4. Steamers
5.10.5. Vibrators

5.11. Hair Massage

5.11.1. Application Techniques

5.12. Treating Cancer Patients

5.12.1. Quality of Life of Cancer Patients and Aesthetic Hair Medicine The Healing Power of Image
5.12.2. Tests Before Procedures in Cancer Patients
5.12.3. Intervention of the Aesthetic Practitioner Before, During, and After Oncological Treatment
5.12.4. Cancer Patient Micronutrition

Module 6. Medical/Pharmacological Treatments and Research in Trichology and New Treatment Alternatives

6.1. Oral Minoxidi (Rogaine®) vs. Topical Minoxidil

6.1.1. Antihypertensive
6.1.2. Available in 2% and 5% Solutions
6.1.3. Desired Effects: Vasodilation, Angiogenesis, and Enhanced Cell Proliferation
6.1.4. Side Effects: Contact Dermatitis and Temporary Hair Loss during the First Four Months of Use
6.1.5. Minoxidil 5% Foam Does Not Contain Propylene Glycol (potential irritant) and Lowers the Incidence of Pruritus

6.2. Oral Dutasteride (Avodart ®): Effectiveness and Safety

6.2.1. Dihydrotestosterone Production Inhibitor for the Treatment of Benign Prostatic Hyperplasia (BPH)
6.2.2. Dutasteride efficacy at 2.5 mg/day
6.2.3. Side Effects

6.3. Finasteride (Propecia®): Most Common for Male Pattern Baldness

6.3.1. A Reductase Inhibitor that Reduces the Conversion of Testosterone to Dihydrotestosterone or DHT Better than Finasteride
6.3.2. More Effective Equal Safety for Men and Women
6.3.3. Women: Avoid Pregnancy during Treatment and 6 Months After. It Is Not Officially Approved for Use in Women

6.3.3.1. Combination with an Effective Oral Contraceptive

6.3.4. Finasteride vs Dutasteride Safety
6.3.5. Dutasteride Microinjections
6.3.6. Improved Hair Quantity and Thickness
6.3.7. Progress Over Time: 6 Months to 1 Year
6.3.8. Daily Dose: 1mg
6.3.9. Problems of a Sexual Nature

6.4. 2% Ketoconazole Topical Shampoo (Nizoral ®)

6.4.1. Antifungal Agent
6.4.2. Treatment for Dermatitis and Dandruff
6.4.3. Action on Scalp Microflora
6.4.4. Beneficial Effect on Androgenetic Alopecia Associated with Hair Follicle Inflammation

6.5. Dexamethasone Minipulse Therapy (alopecia areata): Risks vs. Continuous Corticosteroids
6.6. JAK Inhibitor Drugs (Alopecia Areata)

6.6.1. Clinical Trials: Ruxolitinib or Tofacitinib (Extensive Alopecia Areata)
6.6.2. Efficacy and Safety Results

6.7. Anti-Androgens (Frontal Fibrosing Alopecia): Finasteride and Oral Dutasteride in Females with Frontal Fibrosing Alopecia

6.7.1. Loss of Eyebrows and Hair in the Frontal and Temporal Region (“headband area”)
6.7.2. Block the Binding of Androgen Receptor to Testosterone
6.7.3. Cyproterone Acetate and Spironolactone

6.8. Prostaglandin Analogs

6.8.1. Lipidic Substances Derived from 20-Carbon Fatty Acids (Eicosanoids)
6.8.2. They Affect and Act on Different Systems of the Organism: Nervous System, Smooth Muscle, Blood, and Reproductive System
6.8.3. They Regulate Various Functions: Blood Pressure, Blood Clotting, Allergic Inflammatory Response and Digestive System Activity
6.8.4. Bimatoprost (Latisse®) Is Now Available as a Treatment for Eyelash Growth
6.8.5. Latanoprost Increases Hair Density and May Increase Pigmentation

6.9. Estrogens

6.9.1. Indirect Anti-Androgens

6.10. Hair Cloning

6.10.1. Future Therapies. Unlimited Follicles from Hair Stem Cells. Human Clinical Trials
6.10.2. Two Main Approaches under Investigation: Direct Injection of Cultured Cells or Use of Factors that Promote Cell Multiplication
6.10.3. Cells Are Cultured and the Culture Supernatant Is Processed to Produce a Rich Hair Growth Promoting Compound
6.10.4. PRP: Biostimulation Techniques

6.10.4.1. Increase the Number of Blood Vessels and Improve Circulation
6.10.4.2. Promote the Production of Collagen
6.10.4.3. Counteract the Negative Effect of Free Radicals and Prevent Cellular Aging

6.11. Hair Transplantation with Micrografts in Men and Women

6.11.1. Robots Capable of Automated Extraction

6.12. Low-Power Laser 

6.12.1. Different Wavelengths and Different Modes
6.12.2. Low-Level Laser Intensive (LLLI) Laser Therapy
6.12.3. Uses: Female Androgenetic Alopecia and/or MAGA Male Androgenetic Alopecia Treatments in Monotherapy or as Combined Therapies
6.12.4. Penetrates the Surface of the Skin. Stimulates Blood Flow. Helps Nutrients, Blood, and Oxygen Reach Hair Follicles
6.12.5. Hair Revitalization, Elimination of Toxins and any Blockages Found within the Follicle

6.13. Alternative Treatments 

6.13.1. Herbs, Vitamins, and Minerals
6.13.2. Biotin, Caffeine, Melatonin, Copper Complexes

6.14. Considerations 

6.14.1. Minoxidil and Finasteride Must Be Used Continuously for Results and Once Discontinued, the Natural Baldness Process Resumes
6.14.2. PG Analogs Have a Much More Potent and Longer Lasting Effect, Although Not Permanent
6.14.3. Prostaglandin F2 Alpha Analogs Latanoprost and Bimatoprost Are Used in the Treatment of Ocular Hypertension and Glaucoma

Module 7. Hair Transplantation with the FUSS Technique

7.1. Concept/Definition

7.1.1. History & evolution

7.2. Safe Area Definition
7.3. Advantages
7.4. Disadvantages

7.4.1. Scar
7.4.2. Post-Operative
7.4.3. Suture

7.5. Indications
7.6. Contraindications

7.6.1. Keloids
7.6.2. Black Race

7.7. Technical Aspects

7.7.1. Dissection
7.7.2. Trichophytic Closure

7.8. Post-Operative
7.9. Complications

7.9.1. During Extraction Undermining
7.9.2. After Extraction: Bruising, Pain, Necrosis

7.9.2.1. Treating Complications

Module 8. Hair Transplantation with the FUE Technique

8.1. Hair Micrograft. Concept. Theory. History & Evolution
8.2. Indications for Hair Transplantation
8.3. Contraindications of Hair Transplant
8.4. Advantages and Disadvantages of the FUE Technique

8.4.1. Current Status of the FUE Technique

8.5. Anesthesia of the Donor and Recipient Region
8.6. Allergic Reactions and Anaphylactic Shock
8.7. FUE Technique in Hair Implantology

8.7.1. Choice of Follicular Units
8.7.2. Instruments Used in the FUE Technique
8.7.3. Patient Design
8.7.4. Preparation of the Patient and Donor Site
8.7.5. Extracting Follicular Units
8.7.6. Follicular Unit Maintenance Solutions
8.7.7. Preparing the Receptor Site
8.7.8. Incisions
8.7.9. Implementation

8.8. Implantation with Implanters
8.9. FUE Technique Complications

8.9.1. Intra-Operative
8.9.2. Postoperative

Module 9. Effluvia

9.1. Concept of Effluvium
9.2. Epidemiology
9.3. Effluvia Classification
9.4. Guided Clinical History
9.5. Acute Anagen Effluvium

9.5.1. Pathophysiology of Acute Anagen Effluvium
9.5.2. Diagnosis of Acute Anagen Effluvium

9.5.2.1. Types of Acute Anagen Effluvium
9.5.2.2. Chemotherapy-Induced Dystrophic Effluvium
9.5.2.3. Radiotherapy-Induced Dystrophic Effluvium
9.5.2.4. Toxin-Induced Dystrophic Effluvium

9.6. Chronic Anagen Effluvium

9.6.1. Pathophysiology of Chronic Anagen Effluvium
9.6.2. Diagnosis of Chronic Anagen Effluvium

9.7. Acute Telogen Effluvium

9.7.1. Pathophysiology of Acute Telogen Effluvium
9.7.2. Diagnosis of Acute Telogen Effluvium
9.7.3. Types of Acute Telogen Effluvium

9.8. Chronic Telogen Effluvium

9.8.1. Pathophysiology of Chronic Telogen Effluvium
9.8.2. Diagnosis of Chronic Telogen Effluvium

9.9. Differential Diagnosis of Chronic Telogen Effluvium
9.10. Effluvia Treatment
9.11. Algorithm for Managing Patients with Diffuse Capillary Leakage

Module 10. Legal, Economic, and Marketing Aspects

10.1. Introduction to the Legal Regulations of Professional Development
10.2. Medicolegal Aspects in the Practice of Trichology

10.2.1. Civil and Health Liability

10.3. Legal and Economic Aspects of Free Exercise, Contracting Regimes, Personal Income Tax, VAT, etc.
10.4. Patient-Doctor Relationship

10.4.1. Informed Consent in Capillary Medicine and Surgery
10.4.2. Data Protection, Medical Records Management and Archiving, Iconography (Acquisition and Archiving)
10.4.3. Regulations in Relation to Patients

10.5. Management of a Hair Transplantation and Capillary Medicine Practice

10.5.1. Regulations Regarding Human Resources
10.5.2. Managing Complaints

10.6. Communication Skills in Hair Transplantation and Capillary Medicine
10.7. Media Communication
10.8. Interprofessional Communications

10.8.1. Ethical Principles

10.9. Planning of a Hair Transplantation and a Capillary Medicine Unit
10.10. Organization and Marketing. Sales Techniques for Capillary Surgeons
10.11. Social Networks: Importance and Proper Use

An online program that will allow you to delve into the main sales techniques for the hair surgeon"