TECH offers you the most complete academic program to update your practice in relation to androgenetic diseases, effluvium and alopecia in a 100% online way’’

IAndrogenetic alopecia is one of the most common hair pathologies in both men and women, accounting for around 50% and 40%, respectively, of the world's population. It is a disease that, although it is usually associated with advanced age, can also occur in young patients, affecting their appearance and, therefore, their self-esteem, especially in female cases. The same occurs with telogen effluvium, the alteration of the hair growth cycle that causes a drastic and diffuse hair loss, considerably affecting the amount of hair remaining on the scalp. Like these, there are dozens of hair-related conditions and their frequent presence in society has motivated specific studies to contribute to the establishment of increasingly effective and accurate treatments for each of them.  

It is precisely in this field that TECH, together with a team of specialists in the field of Hair Dermatology, has developed the Postgraduate diploma. It is an academic program of the highest level distributed in 450 hours of the best theoretical, practical and additional content, which has been designed based on the latest and most comprehensive information in the medical sector. Therefore, during the 6 months of the program, the graduate will be able to delve into the latest advances in hair diseases, focusing on Androgenic Alopecia and Effluvium, their early diagnostic strategies and the most effective therapeutic techniques for both men and women.  

All this 100% online through a state-of-the-art Virtual Campus which can be accessed from any device with an internet connection, whether it is a PC, tablet or cell phone. In addition to the syllabus, you will find dozens of hours of additional material (detailed videos, research articles, complementary readings, self-knowledge exercises, news, dynamic summaries, etc.) to delve into the different sections of the program in a personalized way. In addition, you will be able to download it for consultation, even after the academic program is over. This way you will be able to keep up to date on the latest developments in Hair Medicine in an exhaustive and guaranteed way, without worrying about having to attend face-to-face classes or following strict schedules.  

A pathology that delves into the novelties of the pathologies of the scalp over 450 hours of the best theoretical, practical and additional content’’

This Postgraduate diploma in Hair Transplantation Capillary Diseases. Androgenetic Alopecia and Effluvium contains the most complete and up-to-date scientific program on the market. The most important features include: 

• Practical cases presented by experts in Dermatology and Hair Transplantation 
• The graphic, schematic, and practical contents with which they are created, provide scientific and practical information on the disciplines that are essential for professional practice
• Practical exercises where self-assessment can be used to improve learning
• Its special emphasis on innovative methodologies
• Theoretical lessons, questions to the expert, debate forums on controversial topics, and individual reflection assignments 
• Content that is accessible from any fixed or portable device with an Internet connection 

Delving into the complications that can arise in different hair pathologies will help you to implement the latest clinical management strategies to avoid and solve them in your practice’’

The program’s teaching staff includes professionals from sector who contribute their work experience to this program, as well as renowned specialists from leading societies and prestigious universities.  

The multimedia content, developed with the latest educational technology, will provide the professional with situated and contextual learning, i.e., a simulated environment that will provide immersive education programmed to learn in real situations.  

This program is designed around Problem-Based Learning, whereby the professional must try to solve the different professional practice situations that arise during the academic year. For this purpose, the student will be assisted by an innovative interactive video system created by renowned and experienced experts.

You will work intensively on perfecting your diagnostic skills through the most effective and innovative dermatological guidelines"

Thanks to the course of this program, you will be able to address Bromhidrosis chaos based on the curative treatments that are currently having the best results"

Designing the most complete and comprehensive programs is always TECH's priority. Therefore, the graduate who accesses this Postgraduate diploma will find hundreds of hours of the best theoretical, practical and additional content, compacted in a comfortable and accessible curriculum 100% online. In this way, they can work intensively on perfecting their skills, making use of the best and most innovative academic tools in the online university sector. 

Would you like to update your knowledge about trigger foods for hair diseases? In this program you will find a checklist for you to guide your patients through effective diet’’

Module 1 Hair Disorders

1.1. Scalp Pathologies

1.1.1. Dermatosis

1.1.1.1. Dermatoses Affecting the Scalp

1.1.1.1.1. Seborrheic Dermatitis

1.1.1.1.1.1. Description and Origin
1.1.1.1.1.2. Phases of Seborrheic Dermatitis

1.1.1.2. Contact Dermatitis

1.1.1.2.1. Contact Irritant

1.1.1.2.1.1. Chemical Contact
1.1.1.2.1.2. Physical Contact (Allergens)

1.1.1.2.2. Photocontact or Photosensitive

1.1.1.2.2.1. Phototoxic
1.1.1.2.2.2. Photoallergic

1.1.1.3. Erosive-Pustular Dermatosis

1.1.2. Pityriasis

1.1.2.1. Pityriasis
1.1.2.2. Cosmetic Pityriasis
1.1.2.3. Pityriasis Simplex Capitis
1.1.2.4. Steatoid Pityriasis

1.1.3. Scalp Infections and Infestations

1.1.3.1. Superficial Folliculitis (Ostiofolliculitis)
1.1.3.2. Deep Folliculitis (Furunculosis and Carbuncles)

1.1.3.2.1. Folliculitis Decalvans

1.1.3.3. Keloid Folliculitis (Keloid Acne)
1.1.3.4. Candidiasis Folliculitis

1.1.4. Tinea Capitis

1.1.4.1. Non-Inflammatory Tinea (Anthropophilic Dermatophytes)
1.1.4.2. Inflammatory Tinea (By Zoophoric Dermatophytes)

1.1.5. Seborrheic Dermatoses, Description and Types

1.1.5.1. Real Seborrhea
1.1.5.2. Two-Layer Seborrhea
1.1.5.3. Apparent Seborrhea
1.1.5.4. Four-Layer Seborrhea
1.1.5.5. Lichen Planopilaris
1.1.5.6. Pediculosis
1.1.5.7. Capillary Psoriasis

1.1.5.7.1. Exclusive Capillary Involvement: Seborrheic Psoriasis
1.1.5.7.2. Plaques: Types

1.1.5.7.2.1. Isolated
1.1.5.7.2.2. Dispersed
1.1.5.7.2.3. Scarce

1.2. Bromhidrosis

1.2.1. Definition 
1.2.2. Causes

1.2.2.1. Apocrine Sweating
1.2.2.2. Eccrine Sweating

1.2.3. Trigger Foods

1.2.3.1. Other Triggers

1.2.4. Symptoms
1.2.5. Diagnosis
1.2.6. Treatment

1.2.6.1. Botox
1.2.6.2. Liposuction
1.2.6.3. Surgery
1.2.6.4. Home Remedies

1.2.7. Complications

1.2.7.1. Trichomycosis
1.2.7.2. Erythrasma
1.2.7.3. Intertrigo
1.2.7.4. Diabetes Mellitus Type II
1.2.7.5. Obesity

1.3. Congenital Alopecia

1.3.1. Universal
1.3.2. Hereditary Hypotrichosis: Marie-Unna Type
1.3.3. Unclassified Types

1.3.3.1. Localized:

1.3.3.1.1. Aplasia
1.3.3.1.2. Skin

1.3.3.2. Triangular Alopecia
1.3.3.3. Congenital Anonychia

1.3.4. Ectodermal Dysplasias

1.3.4.1. Hydrotic
1.3.4.2. Anhydrotic

1.3.5. Syndromes

1.3.5.1. Autosomal Recessive Conditions

1.3.5.1.1. Cockayne Syndrome
1.3.5.1.2. Werner Syndrome
1.3.5.1.3. Progeria
1.3.5.1.4. Rothmund Syndrome
1.3.5.1.5. Seckel Syndrome
1.3.5.1.6. Menkes Syndrome
1.3.5.1.7. Marinesco Syndrome
1.3.5.1.8. Conradi Syndrome
1.3.5.1.9. Dyskeratosis Congenita
1.3.5.1.10. Cartilage-Hair Hypoplasia
1.3.5.1.11. Enteropathic Acrodermatitis
1.3.5.1.12. Syndromes: Tricho-Rhino-Phalangeal
1.3.5.1.13. Homocystinuria
1.3.5.1.14. Lamellar Ichthyosis
1.3.5.1.15. Hartnut Disease
1.3.5.1.16. Citrulinemia
1.3.5.1.17. Tricorhinophalangeal Syndrome

1.3.6. Autosomal Dominant Conditions

1.3.6.1. Pachyonychia Congenita
1.3.6.2. Hallermann-Streiff Syndrome
1.3.6.3. Oculo-Dento-Digital Syndrome
1.3.6.4. Treacher-Collins Syndrome
1.3.6.5. Popliteal Membrane Syndrome

1.3.7. AX-Linked Dominant Disorders

1.3.7.1. Digital Orofacial Syndrome
1.3.7.2. Incontinentia Pigmenti
1.3.7.3. Focal Dermal Hypoplasia

1.3.8. AX-Linked Recessive Disorders

1.3.8.1. Keratosis Follicularis Spinulosa Decalvans with Ophiasi

1.3.9. Chromosomal Aberrations

1.3.9.1. Down Syndrome - Trisomy 11
1.3.9.2. Trisomy A

1.4. Scarring Alopecia

1.4.1. Definition
1.4.2. Types

1.4.2.1. Caused by the Body Itself

1.4.2.1.1.1. Genetic Determinants

1.4.2.2. Abnormalities

1.4.2.2.1. Folliculitis Decalvans
1.4.2.2.2. Keloid Acne
1.4.2.2.3. Lupus Erythematosus
1.4.2.2.4. Pustular Dermatosis
1.4.2.2.5. Lichen Planus
1.4.2.2.6. Frontal Fibrosing Alopecia (F.F.A.)
1.4.2.2.7. Some Types of General Alopecia Areata

1.4.2.3. Acquired

1.4.2.3.1. Radiotherapy
1.4.2.3.2. Burns
1.4.2.3.3. Surgical

1.5. Other Classifications of Scarring Alopecia

1.5.1. Lymphocytic Infiltrate

1.5.1.1. Chronic Cutaneous Lupus Erythematosus
1.5.1.2. Follicular Lichen Planus
1.5.1.3. Pseudopelade of Brocq
1.5.1.4. Central Centrifugal Cicatricial Alopecia

1.5.2. Neutrophilic Infiltrate:

1.5.2.1. Folliculitis Decalvans
1.5.2.2. Dissecting Cellulitis/Folliculitis

1.5.3. Mixed Infiltrate:

1.5.3.1. Keloid Acne of the Nape
1.5.3.2. Varioliform Necrotic Acne
1.5.3.3. Erosive Pustular Dermatosis

1.5.4. Non-Specific Infiltrate:

1.5.4.1. Scarring Alopecia in Final Stages

1.6. Non-Scarring Alopecia

1.6.1. Definition
1.6.2. Types

1.6.2.1. Androgenetic Alopecia:
1.6.2.2. Traumatic or External Agent Alopecia

1.6.2.2.1. Trichotillomania
1.6.2.2.2. Caused by Chemical Misuse
1.6.2.2.3. Traction Alopecia

1.6.2.3. Alopecia Areata

1.6.2.3.1. Common Areata
1.6.2.3.2. General Areata

1.6.2.4. Drug and Pharmaceutical-Related Alopecia

1.6.2.4.1. Vitamin A
1.6.2.4.2. Anticoagulants
1.6.2.4.3. Mercury
1.6.2.4.4. Boric Acid
1.6.2.4.5. Beta-Blockers

1.6.2.5. Syphilitic Alopecia

1.6.2.5.1. Description
1.6.2.5.2. Features

1.6.2.6. Alopecia Caused by Systemic Diseases

1.6.2.6.1. Infectious
1.6.2.6.2. Endocrine
1.6.2.6.3. Nutritional Deficiency

1.6.2.7. Effluvia

1.6.3. Histopathological Signs

1.6.3.1. Androgenetic Alopecia

1.6.3.1.1. Hair Follicle Miniaturization
1.6.3.1.2. Sebaceous Pseudohyperplasia

1.6.3.2. Telogen Effluvium

1.6.3.2.1. Predominance of Hair Follicles in Telogen Phase
1.6.3.2.2. Absence of Significant Histopathological Changes

1.6.3.3. Alopecia Areata

1.6.3.3.1. Peri and Intrabulbar Lymphocytic Infiltrate (Honeycomb Hyperpigmentation)
1.6.3.3.2. Several Follicles of the Biopsy in the Same Evolutionary Phase
1.6.3.3.3. Reversal of the Anagen-Telogen Ratio

1.6.3.4. Syphilitic Alopecia

1.6.3.4.1. Abundance of Plasma Cells in the Inflammatory Infiltrate
1.6.3.4.2. Presence of Treponema Pallidum with HI stains

1.6.3.5. Trichotillomania

1.6.3.5.1. Absence of Peribulbar Inflammatory Infiltrate
1.6.3.5.2. Trichomalacia
1.6.3.5.3. Incontinentia Pigmenti
1.6.3.5.4. Intra and Perifollicular Hemorrhages

1.6.3.6. Traction Alopecia

1.6.3.6.1. Similar to Trichotillomania
1.6.3.6.2. Diminution of Terminal Hair Follicles

1.7. Hypertrichosis

1.7.1. General

1.7.1.1. Primary or Congenital

1.7.1.1.1. Universal Hypertrichosis or Ambras Syndrome
1.7.1.1.2. Congenital Hypertrichosis Lanuginosa
1.7.1.1.3. Prepubertal Hypertrichosis
1.7.1.1.4. Acquired Hypertrichosis Lanuginosa

1.7.1.2. Secondary or Acquired

1.7.1.2.1. Caused by Drugs or Medication
1.7.1.2.2. Caused by Systemic Diseases

1.7.2. Localized

1.8. Hirsutism

1.8.1. Ovarian SAHA Syndrome
1.8.2. Adrenal SAHA Syndrome
1.8.3. SAHA Syndrome with Hyperprolactinemia
1.8.4. SOP
1.8.5. Hypophyseal Hirsutism
1.8.6. Drug Use
1.8.7. Liver Diseases

1.9. Hyperhydrosis

1.9.1. Definition
1.9.2. Diagnosis
1.9.3. Causes

1.9.3.1. Primary
1.9.3.2. Diffuse

1.9.4. Treatment

1.9.4.1. Antiperspirants
1.9.4.2. Anticholinergics
1.9.4.3. Iontophoresis
1.9.4.4. Botox
2.9.4.5. Microwave Thermolysis

Module 2. Androgenetic Alopecia

2.1. Features

2.1.1. Evolutionary Development
2.1.2. Physiological or Non-Physiological
2.1.3. Mediated by Two Factors: Genetic and Androgenic

2.2. Evolution

2.2.1. Hamilton for Boys
2.2.2.  Ludwig for Girls

2.3. Pathophysiology

2.3.1. Genetic Receptors of the Male Hormone
2.3.2. An Enzyme the 5alpha-Reductase
2.3.3. DHT

2.4. Men
2.5. Women

2.5.1. Physiology
2.5.2. Hormonal
2.5.3. Genetics
2.5.4. Study of the Hypothalamic-Pituitary-Pituitary-Adrenal-Ovarian Axis

2.6. Consequences
2.7. AGA Study: Inclusion in Therapeutic Algorithm

2.7.1. Clinical History with Oriented Anamnesis
2.7.2. Macro and Micro Exploration with Use of Dermatoscopes and Micro Cameras
2.7.3. Taking Photographs
2.7.4. Traction Test
2.7.5. Trichogram:

2.7.5.1. Optical Microscope: 20-50 Hairs
2.7.5.2. Classification of Growth Phases: Anagen (85%) (1-2%) and Telogen (10-15%)
2.7.5.3. Daily Hair Loss
2.7.5.4. Features

2.7.6. Wood Light
2.7.7. Biopsy
2.7.8. Targeted Analysis
2.7.9. Diagnostic Approach

2.7.9.1. Inclusion in Therapeutic Algorithm: Baldness Prevention

2.7.10. According to Resolution

2.7.10.1. Easily Resolved: Seasonal or Cyclic, Androgenetic (MAGA and FAGA), Menopausal and Senile. Effluvium
2.7.10.2. Potentially Resolvable: Pathogen-Mediated

2.7.10.2.1. Psychogenic due to Stress
2.7.10.2.2. Traction and Trichotillomania
2.7.10.2.3. Deficiency (Dietary, Anemic, Vitamin Deficiency)
2.7.10.2.4. Chronic Effluvium
2.7.10.2.5. Hormonal/Androgenic
2.7.10.2.6. Thyrogenic
2.7.10.2.7. Immunogenic
2.7.10.2.8. Chemotherapy
2.7.10.2.9. Collagenosis
2.7.10.2.10. Areata
2.7.10.2.11. Infectious (Bacterial, Mycotic, Syphilis)
2.7.10.2.12. More Common in Women: Multi-Factorial

2.7.10.3. Difficult to Resolve

2.7.10.3.1. Congenital Cicatricial
2.7.10.3.2. FFA
2.7.10.3.3. Physical
2.7.10.3.4. Infections
2.7.10.3.5. Tumours
2.7.10.3.6. Dermatosis (Lupus, Liquen, Psoriasis, etc)

2.8. Treatment

2.8.1. Cosmetic

2.8.1.1. Cleaning and Hygiene: Appropriate Shampoo
2.8.1.2. Moisturizes, Nourishes and Repairs the Hair Shaft
2.8.1.3. Powders, Dyes, Volumizing Sprays and Special Hairstyles
2.8.1.4. Keratin Microfibers
2.8.1.5. Extensions and Prostheses

2.8.2. Diet: Balanced Diet

2.8.2.1. Amino Acids: L-Cysteine
2.8.2.2. Vitamins: B12, Biotin, Folic Acid, etc. 2.8.2.3. Trace Elements: Zinc, Fe, Se, etc.

2.8.3. Topical

2.8.3.1. Non-Specific

2.8.3.1.1. Shampoos: Antimycotics, Antipsoriatics, Keratolytics, etc.    
2.8.3.1.2. Creams, Lotions, Gels, etc.    
2.8.3.1.3. Corticosteroids, Antibiotics, Seboregulators, etc.

2.8.3.2. Specific

2.8.3.2.1. Lotions or Foams
2.8.3.2.2. Spironolactone 2%
2.8.3.2.3. Canrenone 2%
2.8.3.2.4. Progesterone 0.025%
2.8.3.2.5. 17-alpha-estradiol 0.025-0.05%
2.8.3.2.6. Minoxidil 2-5%
2.8.3.2.7. Ac. Retinoic Acid 0.025-0.05%
2.8.3.2.8. Alpha-Tocopherol Nicotinate 5%

2.8.4. Local

2.8.4.1. Drug Dermoinfiltration

2.8.4.1.1. Roller
2.8.4.4.2. Dermojet
2.8.4.4.3. Hair Mesotherapy
2.8.4.4.4. Carboxytherapy

2.8.4.2. Micropigmentation
2.8.4.3. Biological Therapies PRP and Stem Cells
2.8.4.4. Electrophysical Therapy

2.8.4.4.1. Transportation and Ionization
2.8.4.4.2. Infrared and Low Frequency Lasers

2.8.4.5. Capillary Surgery

2.8.5. Systemic

2.8.5.1. Underlying Pathology

2.8.5.1.1. Anti-Fungals/Antibiotics, Thyroid, Anxiolytics, Corticosteroids

2.8.5.2. Androgenetic (AGA) Factor

2.8.5.2.1. Finasteride
2.8.5.2.2. Dutasteride
2.8.5.2.3. Oral Minoxidil

2.8.5.3. Androgenetic Factors: Antiandrogens

2.8.5.3.1. Central: Cyproterone with/without Estradiol
2.8.5.3.2. Peripheral: Spironolactone
2.8.5.3.3. Adrenal: Prednisone and Deflazacort

2.9. Specific Techniques

2.9.1. Hair Mesotherapy
2.9.2. Hair Micrografts
2.9.3. Biological Therapies

2.9.3.1. Plasma
2.9.3.2. Stem Cells

Module 3. Effluvia

3.1. Concept of Effluvium
3.2. Epidemiology
3.3. Effluvia Classification
3.4. Guided Clinical History
3.5. Acute Anagen Effluvium

3.5.1. Pathophysiology of Acute Anagen Effluvium
3.5.2. Diagnosis of Acute Anagen Effluvium

3.5.2.1. Types of Acute Anagen Effluvium
3.5.2.2. Chemotherapy-Induced Dystrophic Effluvium
3.5.2.3. Radiotherapy-Induced Dystrophic Effluvium
3.5.2.4. Toxin-Induced Dystrophic Effluvium

3.6. Chronic Anagen Effluvium

3.6.1. Pathophysiology of Chronic Anagen Effluvium
3.6.2. Diagnosis of Chronic Anagen Effluvium

3.7. Acute Telogen Effluvium

3.7.1. Pathophysiology of Acute Telogen Effluvium
3.7.2. Diagnosis of Acute Telogen Effluvium
3.7.3. Types of Acute Telogen Effluvium

3.8. Chronic Telogen Effluvium

3.8.1. Pathophysiology of Chronic Telogen Effluvium
3.8.2. Diagnosis of Chronic Telogen Effluvium

3.9. Differential Diagnosis of Chronic Telogen Effluvium}
3.10. Effluvia Treatment
3.11. Algorithm for Managing Patients with Diffuse Capillary Leakage

This will provide key training to advance your career"